Several posts highlight the role of social stress and inflammation, such as references to the longstanding link between social adversity and depression, while others discuss the biological mechanisms involved. The tone varies from curiosity to cautious optimism, emphasizing the importance of this work for advancing mental health research.
AgreementModerate agreement
Most discussions acknowledge the connection between social stress, inflammation, and depression, indicating general agreement with the research's framework.
InterestModerate interest
Participants show moderate curiosity, referencing past studies and emphasizing the relevance of social stressors in mental health.
EngagementModerate engagement
The posts include some detailed comments and reflections on the mechanisms involved, demonstrating a basic but thoughtful level of engagement.
ImpactModerate impact
The discussions suggest that this research may influence understanding and future treatment approaches, as seen in mentions of its significance for mental health and potential interventions.
Abstract in brief
Social stressors, especially interpersonal rejection, are major risk factors for depression, and this theory explains how social adversity activates biological processes leading to depression.
The theory suggests that social threats increase inflammation via immune system components called proinflammatory cytokines, which can cause depressive behaviors like sadness, fatigue, and social withdrawal.
This inflammation response, evolved for physical danger, can be triggered by modern social stress, linking depression to other health issues like heart disease, arthritis, and obesity, and highlighting new avenues for prevention and treatment by targeting inflammation.]
Negative thoughts can trigger body inflammation and health issues. Chronic stress activates stress hormones like cortisol, leading to overactive immune responses that attack the body, increasing risks of joint pain…
This video critically analyzes claims about stress suppressing the immune system for six hours, highlighting the lack of empirical support. It emphasizes the importance of scientific accuracy and warns against…
RT @myuuko: うつ病の炎症仮説にかんする2014年のレビュー論文。今はもううつ病は炎症反応あるよねーってことで落ち着いてるのかな。/ From stress to inflammation and major depressive disorder: A social…
Oct 24, 2022 · 4 shares · 3,070 followersView post →
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https://doi.org/10.1037/a0035302. Slavich, G. M., & Shields, G. S. (2018). Assessing lifetime stress exposure using the Stress and Adversity Inventory for ...
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A qualitative exploration of how lifetime stressor exposure influences ...
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https://doi.org/10.1037/a0035302. Slavich, G. M., & Shields, G. S. (2018). Assessing lifetime stress exposure using the stress and adversity inventory for.
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https://doi.org/10.1037/a0035302. MURPHY ET AL. | 447. Page 18. Slavich, G. M., & Sacher, J. (2019). Stress, sex hormones, inflammation, and major depressive ...
org/10.1037/a0035302 Shields, G. S., Spahr, C. M., & Slavich, G. M. (2020). Psychosocial interventions and immune system function: A meta-analysis of ...
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from http://dx.doi.org/10.1037/a0035302. Slavich, G. M., Monroe, S. M., & Gotlib, I. H. (2011). Early parental loss and depression history: Associations with ...
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doi: 10.1037/a0035302. 49, Slavich, G. M., Tartter, M. A., Brennan, P. A., & Hammen, C. (2014). Endogenous opioid system influences depressive reactions to ...
doi:10.1037/a0035302. Slavich, G. M., O'Donovan, A., Epel, E. S., & Kemeny, M. E.. (2010). Black sheep get the blues: A psychobiological model of social ...
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