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The discussions reflect strong agreement and deep interest, with references to experimental evidence, critiques of traditional views, and emphasis on ApoB's causal role, which collectively indicate that this publication significantly influences understanding and debate about atherosclerosis mechanisms.
Most discussions agree with the article's assertion that ApoB-rich lipoprotein retention causes atherosclerosis, citing evidence and emphasizing its central role.
The variety of references, mentions of historical human studies, and debates about mechanisms show high engagement and curiosity about the topic.
Numerous posts reference specific studies, mechanisms (like ApoB’s role), and critique or support ideas, indicating deep involvement.
The discussions underscore the importance of this model in understanding and potentially treating cardiovascular disease, suggesting high relevance.
"It's tempting to think of atherosclerosis as a disease of modern lifestyle. Yet it has plagued humans for thousands of years and has been detected in 4000yo Egyptian mummies" beware the story-telling! https://t.co/wDzHOM4EhP
5/ Genetic studies, observational studies and RCT’s unequivocally show that elevated LDL-C, and more specifically ApoB, CAUSES atherosclerosis. Yes this is considered adequate evidence to make a causal claim. https://t.co/T7wvsDfjyH https://t.co/LEmZmN9siu
3/ Firstly, the mechanism by which elevated LDL cholesterol (LDL-C) increases risk of atherosclerotic CVD is by increasing the # of circulating ApoB containing lipoproteins. https://t.co/T7wvsDfjyH
@NathanEqualsOne @DrNadolsky @DaveDixonPharmD @DineshKalra @dmljmd @ethanjweiss You need nothing more than this https://t.co/A3r3DabtM0
The central role of arterial retention of cholesterol-rich apolipoprotein-B-containing lipoproteins in the pathogenesis of atherosclerosis: a triumph of simplicity (2016)
Cardiologist reasoning: our recommendations are pro-oxidant, therefore oxidation can't be harmful. It's not simple, it's simple-minded. "The Central Role of Arterial Retention of Cholesterol-Rich Apolipoprotein-B-Containing Lipoproteins in the Pathogenesis of Atherosclerosis: A https://t.co/qrModakt2z
This video explores why plaque develops in arteries but not in veins. Learn about the role of cholesterol-rich lipoproteins in atherosclerosis and the factors influencing arterial and venous health.
This video discusses the retention hypothesis of early atherogenesis and the role of cholesterol-rich lipoproteins in artery health. It highlights the impact of cholesterol accumulation on vascular health and its…
Higher dietary fiber intake is linked to a lower risk of cardiovascular disease (CVD) and can improve blood pressure and health factors. This video explores the impact of fiber consumption on overall mortality…
@Alicewants2know @MohammedAlo “LDL and other cholesterol-rich, apoB-containing lipoproteins, once they become retained and modified within the arterial wall, cause atherosclerosis.” https://t.co/d5iexXaFOX
RT @TuckerGoodrich: Cardiologist reasoning: our recommendations are pro-oxidant, therefore oxidation can't be harmful. It's not simple, it…
RT @TuckerGoodrich: Cardiologist reasoning: our recommendations are pro-oxidant, therefore oxidation can't be harmful. It's not simple, it…
RT @TuckerGoodrich: Cardiologist reasoning: our recommendations are pro-oxidant, therefore oxidation can't be harmful. It's not simple, it…
Cardiologist reasoning: our recommendations are pro-oxidant, therefore oxidation can't be harmful. It's not simple, it's simple-minded. "The Central Role of Arterial Retention of Cholesterol-Rich Apolipoprotein-B-Containing Lipoproteins in the Pathogenesis of Atherosclerosis: A https://t.co/qrModakt2z
RT @agingdoc1: The central role of arterial retention of cholesterol-rich apolipoprotein-B-containing lipoproteins in the pathogenesis of a…
RT @agingdoc1: The central role of arterial retention of cholesterol-rich apolipoprotein-B-containing lipoproteins in the pathogenesis of a…
RT @agingdoc1: The central role of arterial retention of cholesterol-rich apolipoprotein-B-containing lipoproteins in the pathogenesis of a…
RT @agingdoc1: The central role of arterial retention of cholesterol-rich apolipoprotein-B-containing lipoproteins in the pathogenesis of a…
RT @agingdoc1: The central role of arterial retention of cholesterol-rich apolipoprotein-B-containing lipoproteins in the pathogenesis of a…
RT @agingdoc1: The central role of arterial retention of cholesterol-rich apolipoprotein-B-containing lipoproteins in the pathogenesis of a…
RT @agingdoc1: The central role of arterial retention of cholesterol-rich apolipoprotein-B-containing lipoproteins in the pathogenesis of a…
RT @agingdoc1: The central role of arterial retention of cholesterol-rich apolipoprotein-B-containing lipoproteins in the pathogenesis of a…
The central role of arterial retention of cholesterol-rich apolipoprotein-B-containing lipoproteins in the pathogenesis of atherosclerosis: a triumph of simplicity (2016)
@ifixhearts A Century of Cholesterol and Coronaries: From Plaques to Genes to Statins https://t.co/3u2CIOGa5z The central role of arterial retention of cholesterol-rich apolipoprotein-B-containing lipoproteins in the pathogenesis of atherosclerosis https://t.co/UQsNHcQS7z
@TotalCytopath their composition affects retention https://t.co/wDzHOM4EhP
Here are some of the studies I shared with him… https://t.co/AWL8tiMbyz
RT @NutritionMadeS3: "It's tempting to think of atherosclerosis as a disease of modern lifestyle. Yet it has plagued humans for thousands o…
@PKennedyeMD 1) lowering particle number via diff mechanisms (pharma, diet, genetics etc) lowers risk, raising them raises it. this huge body of evidence disproves the "firefighter" idea 2) transcytosis from the lumen occurs thru SRB-I https://t.co/HhTpIh09Mk https://t.co/wDzHOM4EhP
@giedriusci @dylanarmbruste3 "If there is progression" being the key phrase. This study explains how ApoB is essential in atherosclerosis. No ApoB, no CVD. Best to keep it low. https://t.co/UVbXyuWPOA
@giedriusci No "if" was mentioned or implied. That explanation doesn't hold up and was abandoned for a better explanation from Williams. https://t.co/hxLtDT8dSl https://t.co/0b3spmsBvX
RT @NutritionMadeS3: "It's tempting to think of atherosclerosis as a disease of modern lifestyle. Yet it has plagued humans for thousands o…
RT @NutritionMadeS3: "It's tempting to think of atherosclerosis as a disease of modern lifestyle. Yet it has plagued humans for thousands o…
RT @NutritionMadeS3: "It's tempting to think of atherosclerosis as a disease of modern lifestyle. Yet it has plagued humans for thousands o…
RT @NutritionMadeS3: "It's tempting to think of atherosclerosis as a disease of modern lifestyle. Yet it has plagued humans for thousands o…
The central role of arterial retention of cholesterol-rich apolipoprotein-B-containing lipoproteins in the pathogenesis of atherosclerosis: a triumph of simplicity (2016) https://t.co/7XicMKq7K2
RT @NutritionMadeS3: "It's tempting to think of atherosclerosis as a disease of modern lifestyle. Yet it has plagued humans for thousands o…
RT @NutritionMadeS3: "It's tempting to think of atherosclerosis as a disease of modern lifestyle. Yet it has plagued humans for thousands o…
RT @NutritionMadeS3: "It's tempting to think of atherosclerosis as a disease of modern lifestyle. Yet it has plagued humans for thousands o…
RT @NutritionMadeS3: "It's tempting to think of atherosclerosis as a disease of modern lifestyle. Yet it has plagued humans for thousands o…
RT @NutritionMadeS3: "It's tempting to think of atherosclerosis as a disease of modern lifestyle. Yet it has plagued humans for thousands o…
RT @NutritionMadeS3: "It's tempting to think of atherosclerosis as a disease of modern lifestyle. Yet it has plagued humans for thousands o…
RT @NutritionMadeS3: "It's tempting to think of atherosclerosis as a disease of modern lifestyle. Yet it has plagued humans for thousands o…
RT @NutritionMadeS3: "It's tempting to think of atherosclerosis as a disease of modern lifestyle. Yet it has plagued humans for thousands o…
"It's tempting to think of atherosclerosis as a disease of modern lifestyle. Yet it has plagued humans for thousands of years and has been detected in 4000yo Egyptian mummies" beware the story-telling! https://t.co/wDzHOM4EhP
RT @Plant_proof: 3/ Firstly, the mechanism by which elevated LDL cholesterol (LDL-C) increases risk of atherosclerotic CVD is by increasing…
RT @Plant_proof: 3/ Firstly, the mechanism by which elevated LDL cholesterol (LDL-C) increases risk of atherosclerotic CVD is by increasing…
RT @Plant_proof: 5/ Genetic studies, observational studies and RCT’s unequivocally show that elevated LDL-C, and more specifically ApoB, CA…
5/ Genetic studies, observational studies and RCT’s unequivocally show that elevated LDL-C, and more specifically ApoB, CAUSES atherosclerosis. Yes this is considered adequate evidence to make a causal claim. https://t.co/T7wvsDfjyH https://t.co/LEmZmN9siu
3/ Firstly, the mechanism by which elevated LDL cholesterol (LDL-C) increases risk of atherosclerotic CVD is by increasing the # of circulating ApoB containing lipoproteins. https://t.co/T7wvsDfjyH
RT @Drlipid: @NathanEqualsOne @DrNadolsky @DaveDixonPharmD @DineshKalra @dmljmd @ethanjweiss You need nothing more than this https://t.co/…
RT @dylanarmbruste3: There is a causal association for LDL and ASCVD full stop. Evidence: Ference et al 2017 doi: 10.1093/eurheartj/ehx144…
@Gearoidmuar @Alecsplaining Nope https://t.co/FbSyfrMXuN LDL/ApoB is central and causal. Insulin resistance appears exacerbating. Once again, manage both. Don't see why you have a problem with this
RT @dylanarmbruste3: There is a causal association for LDL and ASCVD full stop. Evidence: Ference et al 2017 doi: 10.1093/eurheartj/ehx144…
There is a causal association for LDL and ASCVD full stop. Evidence: Ference et al 2017 doi: 10.1093/eurheartj/ehx144 Borén et al 2020 https://t.co/5UAIV2EpCq Tabas et al 2007 https://t.co/Us8TwoGFgx Jan Borén, Kevin Jon Williams DOI: 10.1097/MOL.0000000000000330 (1/n) https://t.co/BFO8K99Gu1
@LDLSkeptic @DrKarlNadolsky @moreyraortho @nicknorwitz @ethanjweiss @DrNadolsky @DBelardoMD @Drgarthdavis @Drlipid @scottisaacsmd https://t.co/xoeMcW59ES Williams and Tabas address endothelial damage. ^ More recent publication: doi: 10.1097/MOL.0000000000000330.
RT @Drlipid: @NathanEqualsOne @DrNadolsky @DaveDixonPharmD @DineshKalra @dmljmd @ethanjweiss You need nothing more than this https://t.co/…
RT @Drlipid: @NathanEqualsOne @DrNadolsky @DaveDixonPharmD @DineshKalra @dmljmd @ethanjweiss You need nothing more than this https://t.co/…
@NathanEqualsOne @DrNadolsky @DaveDixonPharmD @DineshKalra @dmljmd @ethanjweiss You need nothing more than this https://t.co/A3r3DabtM0
@thoroughlyconf1 @RChrisCC Thank you for the links to the papers. Might I suggest this article (PMID: 27472409) Open access: The central role of arterial retention of cholesterol-rich apolipoprotein-B-containing lipoproteins in the pathogenesis of atherosclerosis: a triumph of simplicity. Boren & Williams https://t.co/gURUFFHGIS