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The discussions reflect strong agreement on the significance of the article, highlighted by phrases like 'microclots caused by endotoxins' and 'fibrinaloid microclots,' which underline the profound effects of bacterial components on clotting. The tone demonstrates high interest and deep engagement, especially when users reference prior research, molecular details, and potential health impacts, illustrating recognition of the study’s importance.
Most discussions acknowledge the significance of the findings, supporting the idea that bacterial components like LPS can dramatically influence blood clot structure and disease processes.
Posts show high interest, with many referencing the mechanisms, relevance to diseases like strokes, and potential implications for health, indicating strong curiosity.
Comments often delve into specific details about the molecular processes, cite previous research, and discuss experimental results, demonstrating deep involvement.
This collective discussion emphasizes that understanding LPS's role in clot formation could have profound clinical implications, potentially influencing diagnostics and treatments.
@MclartyKrysten @Be_Kinderr @resiapretorius PS - though the difference is that @resiapretorius and I established that the reason for this is the formation of a variant of fibrin that is *amyloid* in character. This explains e.g. its resistance to fibrinolysis https://t.co/T7TIzR4CdQ
https://t.co/vssCfi9EOY Endotoxins again- they keep popping up everywhere! Are fibrid ameloid microclots caused by endothelial damage and spike protein alone or by the existence of endotoxins in vaccines? NO-ONE is testing us for microclots - and these are highly resistent to
@ssayssayssay @HonorUkb @resiapretorius @TurdFur58439807 @ThingsHiddenn Original LPS paper at https://t.co/T7TIzR4CdQ - rest linked via https://t.co/qWBJ2lwvhn.
@longcovidman @qkate Agree; we first showed the anomalous microclotting with bacterial LPS https://t.co/T7TIzR4CdQ and later with LTA for Gram +ves https://t.co/evB7JUnBE6
@janiesaysyay @resiapretorius 2/2 though we didn't recognise the massive amplification by microbial detritus and its creation of amyloid fibrin until 2015/16 or so. https://t.co/T7TIzR4CdQ
@doctorasadkhan @shaun_qureshi @apresj20 Specifically https://t.co/T7TIzR4CdQ. For spike we only went down to 1 ng/ml https://t.co/jZZH9deTTn
Live blood analysis reveals bacteria in blood may contribute to diseases by triggering abnormal clotting and inflammation. Tiny amounts of bacterial lipopolysaccharide can cause significant amyloid-rich clot formation…
Blood test videos reveal bacteria in blood may contribute to diseases like strokes, Alzheimer's, and arthritis. Tiny amounts of bacterial lipopolysaccharide can induce abnormal blood clot formation, potentially…
Blood test analysis reveals bacteria may influence blood clot structure, potentially contributing to inflammatory and amyloid diseases. Tiny amounts of bacterial lipopolysaccharide can significantly alter clot…
Blood is traditionally considered sterile, but recent studies suggest bacteria may influence disease. Tiny amounts of bacterial lipopolysaccharide can dramatically alter blood clot structure, potentially linking…
@SenseReceptor https://t.co/SAbZk2kYx4 The clots removed from ischaemic stroke patients by mechanical thrombectomy are amyloid in nature/2024 {from the references,dive into the unique clots}⬇️ /https://t.co/1tlBj5TG7f 2016 / https://t.co/oQWPZLpGMF Proteins behaving badly.
RT @scottish_vig: https://t.co/vssCfi9EOY Endotoxins again- they keep popping up everywhere! Are fibrid ameloid microclots caused by endot…
RT @scottish_vig: https://t.co/vssCfi9EOY Endotoxins again- they keep popping up everywhere! Are fibrid ameloid microclots caused by endot…
RT @scottish_vig: https://t.co/vssCfi9EOY Endotoxins again- they keep popping up everywhere! Are fibrid ameloid microclots caused by endot…
RT @scottish_vig: https://t.co/vssCfi9EOY Endotoxins again- they keep popping up everywhere! Are fibrid ameloid microclots caused by endot…
RT @scottish_vig: https://t.co/vssCfi9EOY Endotoxins again- they keep popping up everywhere! Are fibrid ameloid microclots caused by endot…
RT @scottish_vig: https://t.co/vssCfi9EOY Endotoxins again- they keep popping up everywhere! Are fibrid ameloid microclots caused by endot…
https://t.co/vssCfi9EOY Endotoxins again- they keep popping up everywhere! Are fibrid ameloid microclots caused by endothelial damage and spike protein alone or by the existence of endotoxins in vaccines? NO-ONE is testing us for microclots - and these are highly resistent to
RT @dbkell: @pierrevnk @DocAhmadMalik Amyloidogenic clotting induced by LPS at https://t.co/T7TIzR5a3o
@pierrevnk @DocAhmadMalik Amyloidogenic clotting induced by LPS at https://t.co/T7TIzR5a3o
細菌細胞壁産物が微量でfibrinaloid微小血栓形成を触媒できる https://t.co/ZRJ7EhDRF4 生体内でLPSとfibrinogen濃度比はLPSによる10の8乘倍を超える分子増幅で凝固が可能 https://t.co/wu0aNeeyep LPSが極めて低量(モル比10の−8乗)存在で fibrinogen凝固が起こり,fibrinのamyloid形成 https://t.co/CalSzeswdT
機械的血栓除去 脳卒中の血栓大部分がamyloid amyloid凝固誘発分子 細菌細胞壁成分 細菌性リポ多糖類(LPS) https://t.co/ZRJ7EhDRF4 https://t.co/wu0aNeeyep コロナspike蛋白 https://t.co/M3ITQztMfW spike蛋白で高度amyloid形成 https://t.co/1d20wy4nND https://t.co/a8ZBLOS8DP https://t.co/4GYg6viKYC
FemtogramsのEndotoxin⇨Amyloid血栓 https://t.co/rzUcuLcyJl
@Mangan150 Yep, 100% https://t.co/wSz1pcCBgH
@resiapretorius @doc4care Lots of papers at https://t.co/bqvsXdku0v, including https://t.co/T7TIzR4CdQ
@MCAS_WTF @resiapretorius @PutrinoLab Other infections incl bacterial can cause microclots. See e.g. https://t.co/T7TIzR4CdQ
@longcovidman @qkate Agree; we first showed the anomalous microclotting with bacterial LPS https://t.co/T7TIzR4CdQ and later with LTA for Gram +ves https://t.co/evB7JUnBE6
RT @dbkell: @ssayssayssay @HonorUkb @resiapretorius @TurdFur58439807 @ThingsHiddenn Original LPS paper at https://t.co/T7TIzR4CdQ - rest li…
@ssayssayssay @HonorUkb @resiapretorius @TurdFur58439807 @ThingsHiddenn Original LPS paper at https://t.co/T7TIzR4CdQ - rest linked via https://t.co/qWBJ2lwvhn.
@Birk_Lab @giovannilandoni @SanRaffaeleMI @MyUniSR @CiceriFabio @AnnalisaRugger1 @FDeCobelli @SRAnesthesiaICU @lorenzodagna @azangrillo @CCRJournal @BellomoRinaldo LPS is very hydrophobic, but in our original work https://t.co/T7TIzR4CdQ just TINY, TINY amounts were necessary to induce the fibrinaloid microclots. I don't know about phagocytosis - maybe just seen as 'self' by mPhi?
@LocalRachel @JesslovesMJK https://t.co/T7TIzR4CdQ
@EleanorSews @tjalcovrv @resiapretorius @microbeminded2 Sorry, better link is https://t.co/T7TIzR4CdQ
RT @dbkell: @tammbrant @ShaneyWright Bah, I sent the preprint link. Real paper at https://t.co/T7TIzR4CdQ
@tammbrant @ShaneyWright Bah, I sent the preprint link. Real paper at https://t.co/T7TIzR4CdQ
RT @dbkell: @janiesaysyay @resiapretorius 2/2 though we didn't recognise the massive amplification by microbial detritus and its creation o…
@janiesaysyay @resiapretorius 2/2 though we didn't recognise the massive amplification by microbial detritus and its creation of amyloid fibrin until 2015/16 or so. https://t.co/T7TIzR4CdQ
@doctorasadkhan @shaun_qureshi @apresj20 Specifically https://t.co/T7TIzR4CdQ. For spike we only went down to 1 ng/ml https://t.co/jZZH9deTTn
(source https://t.co/zfEeY4wpkb)
RT @dbkell: @ales_frost @Be_Kinderr Not implausible. LPS (and the equivalent LTA in Gram-positive bacteria) is about the most potent induce…
@ales_frost @Be_Kinderr Not implausible. LPS (and the equivalent LTA in Gram-positive bacteria) is about the most potent inducer of fibrin amyloid microclots @resiapretorius and I have found. All papers at https://t.co/bqvsXdku0v, starting for this w https://t.co/T7TIzR4CdQ
@MclartyKrysten @Be_Kinderr @resiapretorius PS - though the difference is that @resiapretorius and I established that the reason for this is the formation of a variant of fibrin that is *amyloid* in character. This explains e.g. its resistance to fibrinolysis https://t.co/T7TIzR4CdQ
doi:10.1098/rsif.2016.0539. Frontiers in Medicine | www.frontiersin.org 29 January 2018 | Volume 4 | Article 239. Kenny and Kell Microbes, Semen, and ...
http://dx.doi.org/10.1098/rsif.2016.0539 PMCID: PMC5046953. 45. Townsend L, Fogarty H, Dyer A, Martin-Loeches I, Bannan C, Nadarajan P, Bergin C,. O'Farrelly ...
Journal of the Royal Society Interface DOI: 10.1098/rsif.2016.0539. Topics: BSE and vCJD/; diabetes/; bacteria. Advertisement. Sign up to our ...
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