Publications by authors named "Alexei Diakov"

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Acute activation of human epithelial sodium channel (ENaC) by serum and glucocorticoid inducible kinase 1 (SGK1) requires prior cleavage of the channel's γ-subunit at its proximal cleavage site.
Alexei Diakov, Florian Sure, Alexandr V Ilyaskin, Christoph Korbmacher

Pflugers Arch· August 2025

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Automated patch-clamp recordings for detecting activators and inhibitors of the epithelial sodium channel (ENaC).
Florian Sure, Markus Rapedius, Alexei Diakov, Marko Bertog, Alison Obergrussberger

Pflugers Arch· June 2025

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Correction to: Two adjacent phosphorylation sites in the C-terminus of the channel's α-subunit have opposing effects on epithelial sodium channel (ENaC) activity.
Alexei Diakov, Viatcheslav Nesterov, Anke Dahlmann, Christoph Korbmacher

Pflugers Arch· September 2022

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Two adjacent phosphorylation sites in the C-terminus of the channel's α-subunit have opposing effects on epithelial sodium channel (ENaC) activity.
Alexei Diakov, Viatcheslav Nesterov, Anke Dahlmann, Christoph Korbmacher

Pflugers Arch· July 2022

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Transmembrane serine protease 2 (TMPRSS2) proteolytically activates the epithelial sodium channel (ENaC) by cleaving the channel's γ-subunit.
Florian Sure, Marko Bertog, Sara Afonso, Alexei Diakov, Ralf Rinke

J Biol Chem· June 2022

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Inhibition of the epithelial sodium channel (ENaC) by connexin 30 involves stimulation of clathrin-mediated endocytosis.
Alexandr V Ilyaskin, Christoph Korbmacher, Alexei Diakov

J Biol Chem· September 2021

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The degenerin region of the human bile acid-sensitive ion channel (BASIC) is involved in channel inhibition by calcium and activation by bile acids.
Alexandr V Ilyaskin, Sonja A Kirsch, Rainer A Böckmann, Heinrich Sticht, Christoph Korbmacher, Alexei Diakov

Pflugers Arch· July 2018

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Erratum to: Sensitisation of TRPV4 by PAR is independent of intracellular calcium signalling and can be mediated by the biased agonist neutrophil elastase.
Silvia Sostegni, Alexei Diakov, Peter McIntyre, Nigel Bunnett, Christoph Korbmacher

Pflugers Arch· June 2017

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Bile acids potentiate proton-activated currents in oocytes expressing human acid-sensing ion channel (ASIC1a).
Alexandr V Ilyaskin, Alexei Diakov, Christoph Korbmacher, Silke Haerteis

Physiol Rep· February 2017

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Activation of the Human Epithelial Sodium Channel (ENaC) by Bile Acids Involves the Degenerin Site.
Alexandr V Ilyaskin, Alexei Diakov, Christoph Korbmacher, Silke Haerteis

J Biol Chem· September 2016

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Sensitisation of TRPV4 by PAR2 is independent of intracellular calcium signalling and can be mediated by the biased agonist neutrophil elastase.
Silvia Sostegni, Alexei Diakov, Peter McIntyre, Nigel Bunnett, Christoph Korbmacher

Pflugers Arch· April 2015

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Pharmacological and electrophysiological characterization of the human bile acid-sensitive ion channel (hBASIC).
Cathérine M T Lefèvre, Alexei Diakov, Silke Haerteis, Christoph Korbmacher, Stefan Gründer

Pflugers Arch· February 2014

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Characterization of the mouse ClC-K1/Barttin chloride channel.
Sébastien L'Hoste, Alexei Diakov, Olga Andrini, Mathieu Genete, Laurent Pinelli

Biochim Biophys Acta· November 2013

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A mutation in the β-subunit of ENaC identified in a patient with cystic fibrosis-like symptoms has a gain-of-function effect.
Robert Rauh, Daniel Soell, Silke Haerteis, Alexei Diakov, Viatcheslav Nesterov

Am J Physiol Lung Cell Mol Physiol· January 2013

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Plasmin and chymotrypsin have distinct preferences for channel activating cleavage sites in the γ subunit of the human epithelial sodium channel.
Silke Haerteis, Matteus Krappitz, Alexei Diakov, Annabel Krappitz, Robert Rauh

J Gen Physiol· October 2012

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Atomic force microscopy reveals the architecture of the epithelial sodium channel (ENaC).
Andrew P Stewart, Silke Haerteis, Alexei Diakov, Christoph Korbmacher, J Michael Edwardson

J Biol Chem· September 2011

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Protein kinase B alpha (PKBα) stimulates the epithelial sodium channel (ENaC) heterologously expressed in Xenopus laevis oocytes by two distinct mechanisms.
Alexei Diakov, Viatcheslav Nesterov, Marianna Mokrushina, Robert Rauh, Christoph Korbmacher

Cell Physiol Biochem· April 2011

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A mutation of the epithelial sodium channel associated with atypical cystic fibrosis increases channel open probability and reduces Na+ self inhibition.
Robert Rauh, Alexei Diakov, Anja Tzschoppe, Judit Korbmacher, Abul Kalam Azad

J Physiol· April 2010

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Cholesterol depletion of the plasma membrane prevents activation of the epithelial sodium channel (ENaC) by SGK1.
Bettina Krueger, Silke Haerteis, Limin Yang, Andrea Hartner, Robert Rauh, Alexei Diakov

Cell Physiol Biochem· February 2010

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Functional characterization of a partial loss-of-function mutation of the epithelial sodium channel (ENaC) associated with atypical cystic fibrosis.
Regina Huber, Bettina Krueger, Alexei Diakov, Judit Korbmacher, Silke Haerteis

Cell Physiol Biochem· February 2010

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Cleavage in the {gamma}-subunit of the epithelial sodium channel (ENaC) plays an important role in the proteolytic activation of near-silent channels.
Alexei Diakov, Katarzyna Bera, Marianna Mokrushina, Bettina Krueger, Christoph Korbmacher

J Physiol· October 2008

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A novel pathway of epithelial sodium channel activation involves a serum- and glucocorticoid-inducible kinase consensus motif in the C terminus of the channel's alpha-subunit.
Alexei Diakov, Christoph Korbmacher

J Biol Chem· September 2004

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