Publications by authors named "Ana Leda Longhini"

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Fasting and fasting-mimicking treatment activate SIRT1/LXRα and alleviate diabetes-induced systemic and microvascular dysfunction.
Sandra S Hammer, Cristiano P Vieira, Delaney McFarland, Maximilian Sandler, Yan Levitsky, Tim F Dorweiler, Todd A Lydic, Bright Asare-Bediako, Yvonne Adu-Agyeiwaah, Micheli S Sielski

Diabetologia· July 2021

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Selective LXR agonist DMHCA corrects retinal and bone marrow dysfunction in type 2 diabetes.
Cristiano P Vieira, Seth D Fortmann, Masroor Hossain, Ana Leda Longhini, Sandra S Hammer

JCI Insight· July 2020

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Red blood cells microparticles are associated with hemolysis markers and may contribute to clinical events among sickle cell disease patients.
Oladele Simeon Olatunya, Carolina Lanaro, Ana Leda Longhini, Carla Fernanda Franco Penteado, Kleber Y Fertrin

Ann Hematol· November 2019

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Up-regulation of SPINT2/HAI-2 by Azacytidine in bone marrow mesenchymal stromal cells affects leukemic stem cell survival and adhesion.
Fernanda Marconi Roversi, Nathalia Moreno Cury, Matheus Rodrigues Lopes, Karla Priscila Ferro, João Agostinho Machado-Neto, Ana Leda Longhini

J Cell Mol Med· February 2019

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Reactive oxygen species production triggers green tea-induced anti-leukaemic effects on acute promyelocytic leukaemia model.
Cristiane Okuda Torello, Rodrigo Naoto Shiraishi, Fernanda Isabel Della Via, Tamara Cristina Lopes de Castro, Ana Leda Longhini

Cancer Lett· February 2018

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Hematopoietic defects in response to reduced Arhgap21.
Juliana Xavier-Ferrucio, Lauremília Ricon, Karla Vieira, Ana Leda Longhini, Mariana Lazarini

Stem Cell Res· January 2018

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Hematopoietic cell kinase (HCK) is a potential therapeutic target for dysplastic and leukemic cells due to integration of erythropoietin/PI3K pathway and regulation of erythropoiesis: HCK in erythropoietin/PI3K pathway.
Fernanda Marconi Roversi, Fernando Vieira Pericole, João Agostinho Machado-Neto, Adriana da Silva Santos Duarte, Ana Leda Longhini

Biochim Biophys Acta Mol Basis Dis· February 2017

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CXCR7 is highly expressed in acute lymphoblastic leukemia and potentiates CXCR4 response to CXCL12.
Rita de Cássia Carvalho Melo, Ana Leda Longhini, Carolina Louzão Bigarella, Mariana Ozello Baratti, Fabiola Traina

PLoS One· October 2014

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