Publications by authors named "Christopher Ginter"

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Novel structural features drive DNA binding properties of Cmr, a CRP family protein in TB complex mycobacteria.
Sridevi Ranganathan, Jonah Cheung, Michael Cassidy, Christopher Ginter, Janice D Pata

Nucleic Acids Res· January 2018

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Structures of paraoxon-inhibited human acetylcholinesterase reveal perturbations of the acyl loop and the dimer interface.
Matthew C Franklin, Michael J Rudolph, Christopher Ginter, Michael S Cassidy, Jonah Cheung

Proteins· September 2016

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Structural insights into mis-regulation of protein kinase A in human tumors.
Jonah Cheung, Christopher Ginter, Michael Cassidy, Matthew C Franklin, Michael J Rudolph

Proc Natl Acad Sci U S A· February 2015

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Protein structure. Structure and activity of tryptophan-rich TSPO proteins.
Youzhong Guo, Ravi C Kalathur, Qun Liu, Brian Kloss, Renato Bruni, Christopher Ginter

Science· January 2015

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The iodide-transport-defect-causing mutation R124H: a δ-amino group at position 124 is critical for maturation and trafficking of the Na+/I- symporter.
Viktoriya Paroder, Juan P Nicola, Christopher S Ginter, Nancy Carrasco

J Cell Sci· August 2013

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Chemical catalysis by the translocator protein (18 kDa).
Christopher Ginter, Irene Kiburu, Olga Boudker

Biochemistry· May 2013

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Conformational ensemble of the sodium-coupled aspartate transporter.
Elka R Georgieva, Peter P Borbat, Christopher Ginter, Jack H Freed, Olga Boudker

Nat Struct Mol Biol· February 2013

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Transport mechanism of a bacterial homologue of glutamate transporters.
Nicolas Reyes, Christopher Ginter, Olga Boudker

Nature· December 2009

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Molecular characterization of V59E NIS, a Na+/I- symporter mutant that causes congenital I- transport defect.
Mia D Reed-Tsur, Antonio De la Vieja, Christopher S Ginter, Nancy Carrasco

Endocrinology· June 2008

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Amino acid residues in transmembrane segment IX of the Na+/I- symporter play a role in its Na+ dependence and are critical for transport activity.
Antonio De la Vieja, Mia D Reed, Christopher S Ginter, Nancy Carrasco

J Biol Chem· August 2007

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Molecular analysis of a congenital iodide transport defect: G543E impairs maturation and trafficking of the Na+/I- symporter.
Antonio De la Vieja, Christopher S Ginter, Nancy Carrasco

Mol Endocrinol· November 2005

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The Q267E mutation in the sodium/iodide symporter (NIS) causes congenital iodide transport defect (ITD) by decreasing the NIS turnover number.
Antonio De La Vieja, Christopher S Ginter, Nancy Carrasco

J Cell Sci· February 2004

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The sodium/iodide Symporter (NIS): characterization, regulation, and medical significance.
Orsolya Dohán, Antonio De la Vieja, Viktoriya Paroder, Claudia Riedel, Mona Artani, Christopher S Ginter

Endocr Rev· February 2003

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Na(+)/I(-) symporter activity requires a small and uncharged amino acid residue at position 395.
Orsolya Dohán, M Verónica Gavrielides, Christopher Ginter, L Mario Amzel, Nancy Carrasco

Mol Endocrinol· August 2002

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