Publications by authors named "Daniel Essigke"

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Activation of the alternative complement pathway and its relevance for sodium retention in experimental nephrotic syndrome.
Daniel Essigke, M Zaher Kalo, Lingsi Kong, Matthias Wörn, Mohammad-Khaled Saad

Res Sq· August 2025

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Impact of aldosterone deficiency on the development of diuretic resistance in mice.
Daniel Essigke, M Zaher Kalo, Andrea Janessa, Bernhard N Bohnert, Xiaqing Li

Pflugers Arch· June 2025

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Transmembrane Serine Protease 2 and Proteolytic Activation of the Epithelial Sodium Channel in Mouse Kidney.
Florian Sure, Sara Afonso, Daniel Essigke, Paul Schmidt, M Zaher Kalo

J Am Soc Nephrol· March 2025

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Amiloride versus furosemide for the treatment of edema in patients with nephrotic syndrome: A pilot study (AMILOR).
Anja Schork, Elisabeth Vogel, Bernhard N Bohnert, Daniel Essigke, Matthias Wörn

Acta Physiol (Oxf)· August 2024

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Sodium retention in nephrotic syndrome is independent of the activation of the membrane-anchored serine protease prostasin (CAP1/PRSS8) and its enzymatic activity.
Daniel Essigke, Bernhard N Bohnert, Andrea Janessa, Matthias Wörn, Kingsley Omage

Pflugers Arch· June 2022

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Proteolytic activation of the epithelial sodium channel (ENaC) by factor VII activating protease (FSAP) and its relevance for sodium retention in nephrotic mice.
Ferruh Artunc, Bernhard N Bohnert, Jonas C Schneider, Tobias Staudner, Florian Sure, Daniel Essigke

Pflugers Arch· February 2022

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Proteinuric chronic kidney disease is associated with altered red blood cell lifespan, deformability and metabolism.
Rosi Bissinger, Travis Nemkov, Angelo D'Alessandro, Marijke Grau, Thomas Dietz, Daniel Essigke

Kidney Int· December 2021

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Experimental nephrotic syndrome leads to proteolytic activation of the epithelial Na channel in the mouse kidney.
Bernhard N Bohnert, Daniel Essigke, Andrea Janessa, Jonas C Schneider, Matthias Wörn

Am J Physiol Renal Physiol· October 2021

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Zymogen-locked mutant prostasin (Prss8) leads to incomplete proteolytic activation of the epithelial sodium channel (ENaC) and severely compromises triamterene tolerance in mice.
Daniel Essigke, Alexandr V Ilyaskin, Matthias Wörn, Bernhard N Bohnert, Mengyun Xiao

Acta Physiol (Oxf)· May 2021

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Plasminogen deficiency does not prevent sodium retention in a genetic mouse model of experimental nephrotic syndrome.
Mengyun Xiao, Bernhard N Bohnert, Hande Aypek, Oliver Kretz, Florian Grahammer, Daniel Essigke

Acta Physiol (Oxf)· January 2021

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Urokinase-type plasminogen activator (uPA) is not essential for epithelial sodium channel (ENaC)-mediated sodium retention in experimental nephrotic syndrome.
Bernhard N Bohnert, Sophie Daiminger, Matthias Wörn, Florian Sure, Tobias Staudner, Daniel Essigke

Acta Physiol (Oxf)· December 2019

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