Publications by authors named "Dat P Ha"

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Requirements for nuclear GRP78 transcriptional regulatory activities and interaction with nuclear GRP94.
Ze Liu, Dat P Ha, Liangguang Leo Lin, Ling Qi, Amy S Lee

J Biol Chem· April 2025

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GRP78 inhibitor YUM70 upregulates 4E-BP1 and suppresses c-MYC expression and viability of oncogenic c-MYC tumors.
Vicky Yamamoto, Dat P Ha, Ze Liu, Miller Huang, Soma Samanta

Neoplasia· September 2024

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Targeting stress induction of GRP78 by cardiac glycoside oleandrin dually suppresses cancer and COVID-19.
Dat P Ha, Woo-Jin Shin, Ze Liu, Michael E Doche, Roy Lau, Shannon M Mumenthaler

Cell Biosci· September 2024

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ER chaperone GRP78/BiP translocates to the nucleus under stress and acts as a transcriptional regulator.
Ze Liu, Guanlin Liu, Dat P Ha, Justin Wang, Min Xiong

Proc Natl Acad Sci U S A· August 2023

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GRP78 Inhibitor YUM70 Suppresses SARS-CoV-2 Viral Entry, Spike Protein Production and Ameliorates Lung Damage.
Dat P Ha, Woo-Jin Shin, Juan Carlos Hernandez, Nouri Neamati, Louis Dubeau

Viruses· May 2023

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Targeting GRP78 suppresses oncogenic KRAS protein expression and reduces viability of cancer cells bearing various KRAS mutations.
Dat P Ha, Bo Huang, Han Wang, Daisy Flores Rangel, Richard Van Krieken

Neoplasia· November 2022

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The stress-inducible ER chaperone GRP78/BiP is upregulated during SARS-CoV-2 infection and acts as a pro-viral protein.
Woo-Jin Shin, Dat P Ha, Keigo Machida, Amy S Lee

Nat Commun· November 2022

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Suppression of ER-stress induction of GRP78 as an anti-neoplastic mechanism of the cardiac glycoside Lanatoside C in pancreatic cancer: Lanatoside C suppresses GRP78 stress induction.
Dat P Ha, Yuan-Li Tsai, Amy S Lee

Neoplasia· December 2021

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The chaperone GRP78 is a host auxiliary factor for SARS-CoV-2 and GRP78 depleting antibody blocks viral entry and infection.
Anthony J Carlos, Dat P Ha, Da-Wei Yeh, Richard Van Krieken, Chun-Chih Tseng, Pu Zhang

J Biol Chem· July 2021

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ER residential chaperone GRP78 unconventionally relocalizes to the cell surface via endosomal transport.
Richard Van Krieken, Yuan-Li Tsai, Anthony J Carlos, Dat P Ha, Amy S Lee

Cell Mol Life Sci· June 2021

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Endoplasmic reticulum chaperone GRP78/BiP is critical for mutant Kras-driven lung tumorigenesis.
Daisy Flores Rangel, Louis Dubeau, Ryan Park, Priscilla Chan, Dat P Ha

Oncogene· May 2021

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Insulin-like growth factor 1-receptor signaling stimulates GRP78 expression through the PI3K/AKT/mTOR/ATF4 axis.
Dat P Ha, Amy S Lee

Cell Signal· November 2020

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The stress-inducible molecular chaperone GRP78 as potential therapeutic target for coronavirus infection.
Dat P Ha, Richard Van Krieken, Anthony J Carlos, Amy S Lee

J Infect· September 2020

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Suppression of stress induction of the 78-kilodalton glucose regulated protein (GRP78) in cancer by IT-139, an anti-tumor ruthenium small molecule inhibitor.
Suzanne J Bakewell, Daisy F Rangel, Dat P Ha, Jyothi Sethuraman, Richard Crouse, Emma Hadley

Oncotarget· July 2018

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Endoplasmic reticulum stress activates SRC, relocating chaperones to the cell surface where GRP78/CD109 blocks TGF-β signaling.
Yuan-Li Tsai, Dat P Ha, He Zhao, Anthony J Carlos, Shan Wei

Proc Natl Acad Sci U S A· May 2018

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GRP78 haploinsufficiency suppresses acinar-to-ductal metaplasia, signaling, and mutant -driven pancreatic tumorigenesis in mice.
Jieli Shen, Dat P Ha, Genyuan Zhu, Daisy F Rangel, Agnieszka Kobielak

Proc Natl Acad Sci U S A· May 2017

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