Publications by authors named "Dominik Honold"

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Stabilization of Monomeric Tau Protein by All D-Enantiomeric Peptide Ligands as Therapeutic Strategy for Alzheimer's Disease and Other Tauopathies.
Tim Altendorf, Ian Gering, Beatrix Santiago-Schübel, Selma Aghabashlou Saisan, Gültekin Tamgüney, Dominik Honold

Int J Mol Sci· January 2023

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Development of an α-synuclein fibril and oligomer specific tracer for diagnosis of Parkinson's disease, dementia with Lewy bodies and multiple system atrophy.
Marc Sevenich, Dominik Honold, Antje Willuweit, Janine Kutzsche, Jeannine Mohrlüder

Neurochem Int· December 2022

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Comparison of the Amyloid Load in the Brains of Two Transgenic Alzheimer's Disease Mouse Models Quantified by Florbetaben Positron Emission Tomography.
Antje Willuweit, Michael Schöneck, Sarah Schemmert, Philipp Lohmann, Saskia Bremen, Dominik Honold

Front Neurosci· October 2021

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Sex-Related Motor Deficits in the Tau-P301L Mouse Model.
Luana Cristina Camargo, Dominik Honold, Robert Bauer, N Jon Shah, Karl-Josef Langen

Biomedicines· September 2021

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In Vitro and In Vivo Efficacies of the Linear and the Cyclic Version of an All-d-Enantiomeric Peptide Developed for the Treatment of Alzheimer's Disease.
Sarah Schemmert, Luana Cristina Camargo, Dominik Honold, Ian Gering, Janine Kutzsche

Int J Mol Sci· June 2021

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PEAβ Triggers Cognitive Decline and Amyloid Burden in a Novel Mouse Model of Alzheimer's Disease.
Luana Cristina Camargo, Michael Schöneck, Nivethini Sangarapillai, Dominik Honold, N Jon Shah

Int J Mol Sci· June 2021

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Comprehensive Characterization of the Pyroglutamate Amyloid-β Induced Motor Neurodegenerative Phenotype of TBA2.1 Mice.
Tina Dunkelmann, Sarah Schemmert, Dominik Honold, Kerstin Teichmann, Elke Butzküven

J Alzheimers Dis· August 2019

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In Vitro Potency and Preclinical Pharmacokinetic Comparison of All-D-Enantiomeric Peptides Developed for the Treatment of Alzheimer's Disease.
Elena Schartmann, Sarah Schemmert, Nicole Niemietz, Dominik Honold, Tamar Ziehm

J Alzheimers Dis· July 2019

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Deceleration of the neurodegenerative phenotype in pyroglutamate-Aβ accumulating transgenic mice by oral treatment with the Aβ oligomer eliminating compound RD2.
Sarah Schemmert, Elena Schartmann, Dominik Honold, Christian Zafiu, Tamar Ziehm

Neurobiol Dis· April 2019

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