Publications by authors named "Hind Mehel"

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Fibroblast growth factor 23 decreases PDE4 expression in heart increasing the risk of cardiac arrhythmia; Klotho opposes these effects.
Marta Lindner, Hind Mehel, Amandine David, Christine Leroy, Martine Burtin

Basic Res Cardiol· July 2020

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Cardiac adenylyl cyclase overexpression precipitates and aggravates age-related myocardial dysfunction.
Nathalie Mougenot, Delphine Mika, Gabor Czibik, Elizabeth Marcos, Shariq Abid, Hind Mehel

Cardiovasc Res· October 2019

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Carboxy-terminal fragment of fibroblast growth factor 23 induces heart hypertrophy in sickle cell disease.
Marie Courbebaisse, Hind Mehel, Camille Petit-Hoang, Jean-Antoine Ribeil, Laurent Sabbah

Haematologica· February 2017

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Phosphodiesterase 2 Protects Against Catecholamine-Induced Arrhythmia and Preserves Contractile Function After Myocardial Infarction.
Christiane Vettel, Marta Lindner, Matthias Dewenter, Kristina Lorenz, Constanze Schanbacher, Hind Mehel

Circ Res· January 2017

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Phosphodiesterase 2: anti-adrenergic friend or hypertrophic foe in heart disease?
Michael Wagner, Hind Mehel, Rodolphe Fischmeister, Ali El-Armouche

Naunyn Schmiedebergs Arch Pharmacol· November 2016

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Dominant negative Ras attenuates pathological ventricular remodeling in pressure overload cardiac hypertrophy.
Manuel Ramos-Kuri, Kleopatra Rapti, Hind Mehel, Shihong Zhang, Perundurai S Dhandapany

Biochim Biophys Acta· November 2015

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Cyclic AMP synthesis and hydrolysis in the normal and failing heart.
Aziz Guellich, Hind Mehel, Rodolphe Fischmeister

Pflugers Arch· June 2014

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Interventricular differences in β-adrenergic responses in the canine heart: role of phosphodiesterases.
Cristina E Molina, Daniel M Johnson, Hind Mehel, Roel L H M G Spätjens, Delphine Mika

J Am Heart Assoc· June 2014

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Phosphodiesterase-2 is up-regulated in human failing hearts and blunts β-adrenergic responses in cardiomyocytes.
Hind Mehel, Julius Emons, Christiane Vettel, Katrin Wittköpper, Danilo Seppelt

J Am Coll Cardiol· October 2013

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Phosphoinositide 3-kinase γ protects against catecholamine-induced ventricular arrhythmia through protein kinase A-mediated regulation of distinct phosphodiesterases.
Alessandra Ghigo, Alessia Perino, Hind Mehel, Alexandra Zahradníková, Fulvio Morello

Circulation· October 2012

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