Publications by authors named "Jacob K Files"

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In situ analysis of neuronal injury and neuroinflammation during HIV-1 infection.
Jenna B Honeycutt, Angela Wahl, Jacob K Files, Alexis F League, Barkha J Yadav-Samudrala

Retrovirology· July 2024

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Impact of Chronic HIV Infection on Acute Immune Responses to SARS-CoV-2.
Skye Opsteen, Tim Fram, Jacob K Files, Emily B Levitan, Paul Goepfert

J Acquir Immune Defic Syndr· May 2024

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Messenger RNA Vaccine Technology: Success for SARS-CoV-2 and Prospects for an HIV-1 Vaccine.
Jacob K Files, Paul A Goepfert

Top Antivir Med· April 2024

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The role of immune activation and antigen persistence in acute and long COVID.
Skye Opsteen, Jacob K Files, Tim Fram, Nathan Erdmann

J Investig Med· June 2023

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HLA-II-Associated HIV-1 Adaptation Decreases CD4 T-Cell Responses in HIV-1 Vaccine Recipients.
Jacob K Files, Sarah Sterrett, Sebastian Henostroza, Christopher Fucile, Kevin Maroney

J Virol· September 2022

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Duration of post-COVID-19 symptoms is associated with sustained SARS-CoV-2-specific immune responses.
Jacob K Files, Sanghita Sarkar, Tim R Fram, Sushma Boppana, Sarah Sterrett

JCI Insight· August 2021

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SARS-CoV-2-specific circulating T follicular helper cells correlate with neutralizing antibodies and increase during early convalescence.
Sushma Boppana, Kai Qin, Jacob K Files, Ronnie M Russell, Regina Stoltz

PLoS Pathog· July 2021

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Sustained cellular immune dysregulation in individuals recovering from SARS-CoV-2 infection.
Jacob K Files, Sushma Boppana, Mildred D Perez, Sanghita Sarkar, Kelsey E Lowman

J Clin Invest· January 2021

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SARS-CoV-2-specific peripheral T follicular helper cells correlate with neutralizing antibodies and increase during convalescence.
Sushma Boppana, Kai Qin, Jacob K Files, Ronnie M Russell, Regina Stoltz

medRxiv· October 2020

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Oncogenic role and therapeutic targeting of ABL-class and JAK-STAT activating kinase alterations in Ph-like ALL.
Kathryn G Roberts, Yung-Li Yang, Debbie Payne-Turner, Wenwei Lin, Jacob K Files

Blood Adv· September 2017

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Photobleaching studies reveal that a single amino acid polymorphism is responsible for the differential binding affinities of linker histone subtypes H1.1 and H1.5.
Thomas W Flanagan, Jacob K Files, Kelsey Rose Casano, Eric M George, David T Brown

Biol Open· February 2016

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