Publications by authors named "Jon Reed"

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Unbiased Proteomic Approach Identifies Pathobiological Profiles in the Brains of Preclinical Models of Repetitive Mild Traumatic Brain Injury, Tauopathy, and Amyloidosis.
Joseph O Ojo, Gogce Crynen, Moustafa Algamal, Prashanti Vallabhaneni, Paige Leary, Jon M Reed

ASN Neuro· October 2021

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APOE genotype dependent molecular abnormalities in the cerebrovasculature of Alzheimer's disease and age-matched non-demented brains.
Joseph O Ojo, Jon M Reed, Gogce Crynen, Prashanthi Vallabhaneni, James Evans

Mol Brain· July 2021

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Molecular Pathobiology of the Cerebrovasculature in Aging and in Alzheimers Disease Cases With Cerebral Amyloid Angiopathy.
Joseph O Ojo, Jon M Reed, Gogce Crynen, Prashanthi Vallabhaneni, James Evans

Front Aging Neurosci· May 2021

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Optimization of a transient antibody expression platform towards high titer and efficiency.
Elizabeth Greene, Daniela Cazacu, Ninkka Tamot, Steven Castellano, Akshita Datar, Jon Reed

Biotechnol J· April 2021

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Molecular abnormalities in autopsied brain tissue from the inferior horn of the lateral ventricles of nonagenarians and Alzheimer disease patients.
Andrew Pearson, Rosa Ajoy, Gogce Crynen, Jon M Reed, Moustafa Algamal

BMC Neurol· August 2020

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Correction to: Mild TBI Results in a Long-Term Decrease in Circulating Phospholipids in a Mouse Model of Injury.
Tanja Emmerich, Laila Abdullah, Joseph Ojo, Benoit Mouzon, Thinh Nguyen, Jon Reed

Neuromolecular Med· June 2020

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Proteomic Identification of Pathways Responsible for the Estradiol Therapeutic Window in AD Animal Models.
Jie Cui, Jon Reed, Gogce Crynen, Ghania Ait-Ghezala, Fiona Crawford

Front Cell Neurosci· October 2019

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Unbiased Proteomic Approach Identifies Unique and Coincidental Plasma Biomarkers in Repetitive mTBI and AD Pathogenesis.
Joseph O Ojo, Gogce Crynen, Jon M Reed, Rosa Ajoy, Prashanthi Vallabhaneni

Front Aging Neurosci· December 2018

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Hippocampal Proteomic Analysis Reveals Distinct Pathway Deregulation Profiles at Early and Late Stages in a Rat Model of Alzheimer's-Like Amyloid Pathology.
Sonia Do Carmo, Gogce Crynen, Tiffany Paradis, Jon Reed, M Florencia Iulita

Mol Neurobiol· April 2018

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Complementary proteomic approaches reveal mitochondrial dysfunction, immune and inflammatory dysregulation in a mouse model of Gulf War Illness.
Zuchra Zakirova, Jon Reed, Gogce Crynen, Lauren Horne, Samira Hassan

Proteomics Clin Appl· September 2017

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Mild TBI Results in a Long-Term Decrease in Circulating Phospholipids in a Mouse Model of Injury.
Tanja Emmerich, Laila Abdullah, Joseph Ojo, Benoit Mouzon, Thinh Nguyen, Jon Reed

Neuromolecular Med· March 2017

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An independent validation of a gene expression signature to differentiate malignant melanoma from benign melanocytic nevi.
Loren E Clarke, Darl D Flake, Klaus Busam, Clay Cockerell, Klaus Helm, Jon Reed

Cancer· February 2017

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Translational potential of long-term decreases in mitochondrial lipids in a mouse model of Gulf War Illness.
Laila Abdullah, James E Evans, Utsav Joshi, Gogce Crynen, Jon Reed

Toxicology· November 2016

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Sub-Chronic Neuropathological and Biochemical Changes in Mouse Visual System after Repetitive Mild Traumatic Brain Injury.
Radouil Tzekov, Clint Dawson, Megan Orlando, Benoit Mouzon, Jon Reed

PLoS One· September 2016

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Plasma Lipidomic Profiling in a Military Population of Mild Traumatic Brain Injury and Post-Traumatic Stress Disorder with Apolipoprotein E ɛ4-Dependent Effect.
Tanja Emmerich, Laila Abdullah, Gogce Crynen, Michael Dretsch, James Evans, Jon Reed

J Neurotrauma· July 2016

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Gulf War agent exposure causes impairment of long-term memory formation and neuropathological changes in a mouse model of Gulf War Illness.
Zuchra Zakirova, Miles Tweed, Gogce Crynen, Jon Reed, Laila Abdullah

PLoS One· February 2016

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Lipidomic analyses identify injury-specific phospholipid changes 3 mo after traumatic brain injury.
Laila Abdullah, James E Evans, Scott Ferguson, Benoit Mouzon, Hannah Montague, Jon Reed

FASEB J· December 2014

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Chronic elevation of phosphocholine containing lipids in mice exposed to Gulf War agents pyridostigmine bromide and permethrin.
Laila Abdullah, James E Evans, Hannah Montague, Jon M Reed, Ann Moser

Neurotoxicol Teratol· July 2014

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Exposure to an organophosphate pesticide, individually or in combination with other Gulf War agents, impairs synaptic integrity and neuronal differentiation, and is accompanied by subtle microvascular injury in a mouse model of Gulf War agent exposure.
Joseph O Ojo, Laila Abdullah, James Evans, Jon Mike Reed, Hannah Montague

Neuropathology· April 2014

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Effect of venlafaxine and desvenlafaxine on drug efflux protein expression and biodistribution in vivo.
Corbin Bachmeier, Gary M Levin, David Beaulieu-Abdelahad, Jon Reed, Michael Mullan

J Pharm Sci· October 2013

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Anti-inflammatory activity of anatabine via inhibition of STAT3 phosphorylation.
Daniel Paris, David Beaulieu-Abdelahad, Laila Abdullah, Corbin Bachmeier, Ghania Ait-Ghezala, Jon Reed

Eur J Pharmacol· January 2013

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Lipidomic profiling of phosphocholine-containing brain lipids in mice with sensorimotor deficits and anxiety-like features after exposure to Gulf War agents.
Laila Abdullah, James E Evans, Alex Bishop, Jon M Reed, Gogce Crynen

Neuromolecular Med· December 2012

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Identification of plasma biomarkers of TBI outcome using proteomic approaches in an APOE mouse model.
Fiona Crawford, Gogce Crynen, Jon Reed, Benoit Mouzon, Alex Bishop

J Neurotrauma· January 2012

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Proteomic CNS profile of delayed cognitive impairment in mice exposed to Gulf War agents.
Laila Abdullah, Gogce Crynen, Jon Reed, Alex Bishop, John Phillips

Neuromolecular Med· December 2011

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Anatabine lowers Alzheimer's Aβ production in vitro and in vivo.
Daniel Paris, David Beaulieu-Abdelahad, Corbin Bachmeier, Jon Reed, Ghania Ait-Ghezala

Eur J Pharmacol· November 2011

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