Publications by authors named "Jonathan C Kentish"

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Right Ventricle Has Normal Myofilament Function But Shows Perturbations in the Expression of Extracellular Matrix Genes in Patients With Tetralogy of Fallot Undergoing Pulmonary Valve Replacement.
Daniel Brayson, So-Jin Holohan, Sonya C Bardswell, Matthew Arno, Han Lu, Jonathan C Kentish

J Am Heart Assoc· August 2020

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Monophosphorylation of cardiac troponin-I at Ser-23/24 is sufficient to regulate cardiac myofibrillar Ca sensitivity and calpain-induced proteolysis.
Abel Martin-Garrido, Brandon J Biesiadecki, Hussam E Salhi, Yasin Shaifta, Cristobal G Dos Remedios, Jonathan C Kentish

J Biol Chem· June 2018

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A model of cardiac contraction based on novel measurements of tension development in human cardiomyocytes.
Sander Land, So-Jin Park-Holohan, Nicolas P Smith, Cristobal G Dos Remedios, Jonathan C Kentish

J Mol Cell Cardiol· May 2017

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Reduced First-Phase Ejection Fraction and Sustained Myocardial Wall Stress in Hypertensive Patients With Diastolic Dysfunction: A Manifestation of Impaired Shortening Deactivation That Links Systolic to Diastolic Dysfunction and Preserves Systolic Ejection Fraction.
Haotian Gu, Ye Li, Henry Fok, John Simpson, Jonathan C Kentish

Hypertension· April 2017

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Increased myofilament Ca2+ sensitivity and diastolic dysfunction as early consequences of Mybpc3 mutation in heterozygous knock-in mice.
Bodvaël Fraysse, Florian Weinberger, Sonya C Bardswell, Friederike Cuello, Nicolas Vignier, Jonathan C Kentish

J Mol Cell Cardiol· June 2012

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cMyBP-C as a promiscuous substrate: phosphorylation by non-PKA kinases and its potential significance.
Sonya C Bardswell, Friederike Cuello, Jonathan C Kentish, Metin Avkiran

J Muscle Res Cell Motil· May 2012

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Novel role for p90 ribosomal S6 kinase in the regulation of cardiac myofilament phosphorylation.
Friederike Cuello, Sonya C Bardswell, Robert S Haworth, Elisabeth Ehler, Sakthivel Sadayappan, Jonathan C Kentish

J Biol Chem· February 2011

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Normal passive viscoelasticity but abnormal myofibrillar force generation in human hypertrophic cardiomyopathy.
Anita C Hoskins, Adam Jacques, Sonya C Bardswell, William J McKenna, Victor Tsang, Jonathan C Kentish

J Mol Cell Cardiol· November 2010

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Esmolol cardioplegia: the cellular mechanism of diastolic arrest.
Hazem B Fallouh, Sonya C Bardswell, Linda M McLatchie, Michael J Shattock, David J Chambers, Jonathan C Kentish

Cardiovasc Res· August 2010

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Enhanced length-dependent Ca2+ activation in fish cardiomyocytes permits a large operating range of sarcomere lengths.
Simon M Patrick, Anita C Hoskins, Jonathan C Kentish, Ed White, Holly A Shiels

J Mol Cell Cardiol· May 2010

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Passive stiffness of myocardium from congenital heart disease and implications for diastole.
Rajiv R Chaturvedi, Todd Herron, Robert Simmons, Darryl Shore, Pankaj Kumar, Jonathan C Kentish

Circulation· March 2010

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Distinct sarcomeric substrates are responsible for protein kinase D-mediated regulation of cardiac myofilament Ca2+ sensitivity and cross-bridge cycling.
Sonya C Bardswell, Friederike Cuello, Alexandra J Rowland, Sakthivel Sadayappan, Jeffrey Robbins, Jonathan C Kentish

J Biol Chem· February 2010

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From genotype to phenotype: a longitudinal study of a patient with hypertrophic cardiomyopathy due to a mutation in the MYBPC3 gene.
Adam Jacques, Anita C Hoskins, Jonathan C Kentish, Steven B Marston

J Muscle Res Cell Motil· December 2009

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Functional analysis of a unique troponin c mutation, GLY159ASP, that causes familial dilated cardiomyopathy, studied in explanted heart muscle.
Emma C Dyer, Adam M Jacques, Anita C Hoskins, Douglas G Ward, Clare E Gallon, Jonathan C Kentish

Circ Heart Fail· September 2009

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Protein phosphatase 2A contributes to the cardiac dysfunction induced by endotoxemia.
Melanie Marshall, Narayana Anilkumar, Joanne Layland, Simon J Walker, Jonathan C Kentish

Cardiovasc Res· April 2009

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Targeting for cardioplegia: arresting agents and their safety.
Hazem B Fallouh, Jonathan C Kentish, David J Chambers

Curr Opin Pharmacol· April 2009

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Protein kinase D selectively targets cardiac troponin I and regulates myofilament Ca2+ sensitivity in ventricular myocytes.
Friederike Cuello, Sonya C Bardswell, Robert S Haworth, Xiaoke Yin, Susanne Lutz, Jonathan C Kentish

Circ Res· March 2007

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Oxidant-induced activation of type I protein kinase A is mediated by RI subunit interprotein disulfide bond formation.
Jonathan P Brennan, Sonya C Bardswell, Joseph R Burgoyne, William Fuller, Ewald Schröder, Jonathan C Kentish

J Biol Chem· August 2006

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Activation of myocardial contraction by the N-terminal domains of myosin binding protein-C.
Todd J Herron, Elena Rostkova, Gudrun Kunst, Rajiv Chaturvedi, Mathias Gautel, Jonathan C Kentish

Circ Res· May 2006

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Protection against endotoxemia-induced contractile dysfunction in mice with cardiac-specific expression of slow skeletal troponin I.
Joanne Layland, Alison C Cave, Chad Warren, David J Grieve, Emma Sparks, Jonathan C Kentish

FASEB J· July 2005

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Protein kinase D is a novel mediator of cardiac troponin I phosphorylation and regulates myofilament function.
Robert S Haworth, Friederike Cuello, Todd J Herron, Gereon Franzen, Jonathan C Kentish

Circ Res· November 2004

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Myofilament-based relaxant effect of isoprenaline revealed during work-loop contractions in rat cardiac trabeculae.
Joanne Layland, Jonathan C Kentish

J Physiol· October 2002

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