Publications by authors named "Jonathan Satin"

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RRAD-reduction reveals efficacy of targeting L-type calcium channel regulation for treatment of heart failure.
Garrett Elmore, Sarisha S Lohano, Nicholas M McVay, Bryana M Levitan, Andrea Sebastian, Jonathan Satin

Cardiovasc Res· November 2025

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Neuro293: A REST-knockout HEK-293 cell line enables the expression of neuron-restricted genes for the high-throughput testing of human neurobiology and the biochemistry of neuronal proteins.
Joshua T Moses, Fahad B Shah, Nicholas M McVay, Dylan E Capes, Christopher C Bosse-Joseph, Jonathan Satin

Biol Methods Protoc· May 2025

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Pharmacological or genetic inhibition of LTCC promotes cardiomyocyte proliferation through inhibition of calcineurin activity.
Lynn A C Devilée, Abou Bakr M Salama, Jessica M Miller, Janice D Reid, Qinghui Ou, Jonathan Satin

NPJ Regen Med· January 2025

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The C-terminus of Rad is required for membrane localization and L-type calcium channel regulation.
Garrett Elmore, Brooke M Ahern, Nicholas M McVay, Kyle W Barker, Sarisha S Lohano, Jonathan Satin

J Gen Physiol· September 2024

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Inherent Metabolic Adaptations in Adult Spiny Mouse ( ) Cardiomyocytes Facilitate Enhanced Cardiac Recovery Following Myocardial Infarction.
Annapurna Kuppa, Afnan Alzamrooni, Rachel Lopez, Tahra Suhan, Rajesh Chaudhary, Jonathan Satin

bioRxiv· May 2024

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Pharmacological or genetic inhibition of LTCC promotes cardiomyocyte proliferation through inhibition of calcineurin activity.
Lynn A C Devilée, Jessica M Miller, Janice D Reid, Abou Bakr M Salama, Qinghui Ou, Jonathan Satin

Res Sq· November 2023

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Author Correction: Adult spiny mice (Acomys) exhibit endogenous cardiac recovery in response to myocardial infarction.
Hsuan Peng, Kazuhiro Shindo, Renée R Donahue, Erhe Gao, Brooke M Ahern, Jonathan Satin

NPJ Regen Med· July 2023

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Transient Cell Cycle Induction in Cardiomyocytes to Treat Subacute Ischemic Heart Failure.
Riham R E Abouleisa, Abou Bakr M Salama, Qinghui Ou, Xian-Liang Tang, Mitesh Solanki, Jonathan Satin

Circulation· April 2022

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Adult spiny mice (Acomys) exhibit endogenous cardiac recovery in response to myocardial infarction.
Hsuan Peng, Kazuhiro Shindo, Renée R Donahue, Erhe Gao, Brooke M Ahern, Jonathan Satin

NPJ Regen Med· November 2021

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Editorial: Recent Advances in Cardiotoxicity Testing.
Tamer M A Mohamed, Javid Moslehi, Jonathan Satin

Front Pharmacol· November 2021

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L-type channel inactivation balances the increased peak calcium current due to absence of Rad in cardiomyocytes.
Brooke M Ahern, Andrea Sebastian, Bryana M Levitan, Jensen Goh, Douglas A Andres, Jonathan Satin

J Gen Physiol· September 2021

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Increased Retention of Cardiac Cells to a Glass Substrate through Streptavidin-Biotin Affinity.
Kara A Davis, Jensen Z Goh, Andrea H Sebastian, Brooke M Ahern, Christine A Trinkle, Jonathan Satin

ACS Omega· July 2021

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Rad-GTPase contributes to heart rate via L-type calcium channel regulation.
Bryana M Levitan, Brooke M Ahern, Ajoy Aloysius, Laura Brown, Yuan Wen, Jonathan Satin

J Mol Cell Cardiol· May 2021

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Heart slice culture system reliably demonstrates clinical drug-related cardiotoxicity.
Jessica M Miller, Moustafa H Meki, Qinghui Ou, Sharon A George, Anna Gams, Jonathan Satin

Toxicol Appl Pharmacol· November 2020

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Improved workflow for mass spectrometry-based metabolomics analysis of the heart.
Douglas A Andres, Lyndsay E A Young, Sudhakar Veeranki, Tara R Hawkinson, Bryana M Levitan, Jonathan Satin

J Biol Chem· February 2020

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Myocardial-restricted ablation of the GTPase RAD results in a pro-adaptive heart response in mice.
Brooke M Ahern, Bryana M Levitan, Sudhakar Veeranki, Mihir Shah, Nemat Ali, Jonathan Satin

J Biol Chem· July 2019

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Elevated myonuclear density during skeletal muscle hypertrophy in response to training is reversed during detraining.
Cory M Dungan, Kevin A Murach, Kaitlyn K Frick, Savannah R Jones, Samuel E Crow, Jonathan Satin

Am J Physiol Cell Physiol· May 2019

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The L-type calcium channel current modulation mechanism: the plot thickens and fogs.
Brooke M Ahern, Jonathan Satin

J Clin Invest· February 2019

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Rad GTPase deletion attenuates post-ischemic cardiac dysfunction and remodeling.
Janet R Manning, Lakshman Chelvarajan, Bryana M Levitan, Catherine N Withers, Prabhakara R Nagareddy, Jonathan Satin

JACC Basic Transl Sci· February 2018

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Rad GTPase is essential for the regulation of bone density and bone marrow adipose tissue in mice.
Catherine N Withers, Drew M Brown, Innocent Byiringiro, Matthew R Allen, Keith W Condon, Jonathan Satin

Bone· October 2017

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Rad-deletion Phenocopies Tonic Sympathetic Stimulation of the Heart.
Bryana M Levitan, Janet R Manning, Catherine N Withers, Jeffrey D Smith, Robin M Shaw, Jonathan Satin

J Cardiovasc Transl Res· December 2016

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Loss of Rad-GTPase produces a novel adaptive cardiac phenotype resistant to systolic decline with aging.
Janet R Manning, Catherine N Withers, Bryana Levitan, Jeffrey D Smith, Douglas A Andres, Jonathan Satin

Am J Physiol Heart Circ Physiol· October 2015

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Role of gap junction channel in the development of beat-to-beat action potential repolarization variability and arrhythmias.
Janos Magyar, Tamas Banyasz, Norbert Szentandrassy, Kornel Kistamas, Peter P Nanasi, Jonathan Satin

Curr Pharm Des· September 2015

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Arrhythmogenic calmodulin mutations disrupt intracellular cardiomyocyte Ca2+ regulation by distinct mechanisms.
Guo Yin, Faisal Hassan, Ayman R Haroun, Lisa L Murphy, Lia Crotti, Jonathan Satin

J Am Heart Assoc· June 2014

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Rad GTPase deletion increases L-type calcium channel current leading to increased cardiac contraction.
Janet R Manning, Guo Yin, Catherine N Kaminski, Janos Magyar, Han-Zhong Feng, Jonathan Satin

J Am Heart Assoc· December 2013

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