Publications by authors named "Julia Staab"

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N-terminal truncation of STAT1 transcription factor causes CD3- and CD20-negative non-Hodgkin lymphoma through upregulation of STAT3-mediated oncogenic functions.
Sana Mumtaz Sheikh, Julia Staab, Martina Bleyer, Aleksandar Ivetic, Fred Lühder

Cell Commun Signal· April 2025

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Social inhibition in depressed patients is associated with an altered activation profile of the interleukin-6-inducible transcription factor STAT3.
Katharina von Knebel, Julia Staab, Anke Gregus, Linus Remling, Oliver Wirths

Brain Behav Immun Health· March 2025

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Associations of attention-deficit/hyperactivity disorder with inflammatory diseases. Results from the nationwide German Health Interview and Examination Survey for Children and Adolescents (KiGGS).
Lena Boemanns, Julia Staab, Thomas Meyer

Neuropsychiatr· December 2024

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A randomized vagus nerve stimulation study demonstrates that serum aldosterone levels decrease with age in women, but not in men.
Elisabeth Veiz, Susann-Kristin Kieslich, Dirk Czesnik, Christoph Herrmann-Lingen, Thomas Meyer, Julia Staab

Sci Rep· August 2023

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A novel interface between the N-terminal and coiled-coil domain of STAT1 functions in an auto-inhibitory manner.
Linus Remling, Anke Gregus, Oliver Wirths, Thomas Meyer, Julia Staab

Cell Commun Signal· July 2023

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Association between self-rated depressive symptoms and mucosal expression of NF-κ B in patients with upper gastrointestinal symptoms.
Julia Staab, Lara Vonhören, Harald Schwörer, Thomas Meyer

Biopsychosoc Med· February 2023

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Lack of STAT1 co-operative DNA binding protects against adverse cardiac remodelling in acute myocardial infarction.
Asmma Doudin, Theresa Riebeling, Julia Staab, Priyanka Rajeev Menon, Fred Lühder

Front Cardiovasc Med· February 2023

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Increased Concentrations of Circulating Interleukins following Non-Invasive Vagus Nerve Stimulation: Results from a Randomized, Sham-Controlled, Crossover Study in Healthy Subjects.
Elisabeth Veiz, Susann-Kristin Kieslich, Dirk Czesnik, Christoph Herrmann-Lingen, Thomas Meyer, Julia Staab

Neuroimmunomodulation· December 2022

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Feasibility and preliminary efficacy of perioperative interventions in patients undergoing coronary artery bypass grafting: the randomized controlled I-COPE trial.
Monika Sadlonova, Jonas Nagel, Svenja Becker, Sophie Neumann, Julia Staab

Eur J Cardiothorac Surg· June 2022

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Evaluation of the putative lymphoma-associated point mutation D427H in the STAT3 transcription factor.
Lena Sophie Behrendsen, Priyanka Rajeev Menon, Muhammad Jawad Khan, Anke Gregus, Oliver Wirths, Julia Staab

BMC Mol Cell Biol· June 2022

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An inhibitory effect on the nuclear accumulation of phospho-STAT1 by its unphosphorylated form.
Priyanka Rajeev Menon, Julia Staab, Anke Gregus, Oliver Wirths, Thomas Meyer

Cell Commun Signal· March 2022

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The anti-parallel dimer binding interface in STAT3 transcription factor is required for the inactivation of cytokine-mediated signal transduction.
Priyanka Rajeev Menon, Asmma Doudin, Anke Gregus, Oliver Wirths, Julia Staab

Biochim Biophys Acta Mol Cell Res· November 2021

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Men Show Reduced Cardiac Baroreceptor Sensitivity during Modestly Painful Electrical Stimulation of the Forearm: Exploratory Results from a Sham-Controlled Crossover Vagus Nerve Stimulation Study.
Elisabeth Veiz, Susann-Kristin Kieslich, Julia Staab, Dirk Czesnik, Christoph Herrmann-Lingen

Int J Environ Res Public Health· October 2021

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Headache Is Associated with Low Systolic Blood Pressure and Psychosocial Problems in German Adolescents: Results from the Population-Based German KiGGS Study.
Melissa A Centeno Córdova, Daniela Stausberg, Biyao Wang, Andreas Becker, Aribert Rothenberger, Julia Staab

J Clin Med· April 2021

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The pathogenic T387A missense mutation in the gene encoding signal transducer and activator of transcription 1 exhibits a differential gene expression profile.
Julia Staab, Till Schwämmle, Thomas Meyer

Mol Immunol· December 2020

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Length Polymorphisms in the Angiotensin I-Converting Enzyme Gene and the Serotonin-Transporter-Linked Polymorphic Region Constitute a Risk Haplotype for Depression in Patients with Coronary Artery Disease.
Thomas Meyer, Isabel Rothe, Julia Staab, Hans-Christian Deter, Stella V Fangauf

Biochem Genet· August 2020

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Identification of a distinct subset of disease-associated gain-of-function missense mutations in the STAT1 coiled-coil domain as system mutants.
Jana Petersen, Julia Staab, Oliver Bader, Timo Buhl, Aleksandar Ivetic

Mol Immunol· October 2019

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Toxoplasma gondii stabilises tetrameric complexes of tyrosine-phosphorylated signal transducer and activator of transcription-1 and leads to its sustained and promiscuous DNA binding.
Roswitha Nast, Julia Staab, Thomas Meyer, Carsten G K Lüder

Cell Microbiol· November 2018

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Do the two transcription factors form a positive feedback amplifier circuit in their common fight against pathogens?
Julia Staab, Roswitha Nast, Thomas Meyer

Immunol Cell Biol· November 2018

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Common variable immunodeficiency, impaired neurological development and reduced numbers of T regulatory cells in a 10-year-old boy with a STAT1 gain-of-function mutation.
Robin Kobbe, Manuela Kolster, Sebastian Fuchs, Ulf Schulze-Sturm, Jutta Jenderny, Julia Staab

Gene· July 2016

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The two interfaces of the STAT1 N-terminus exhibit opposite functions in IFNγ-regulated gene expression.
Julia Staab, Theresa Riebeling, Verena Koch, Christoph Herrmann-Lingen, Thomas Meyer

Mol Immunol· October 2015

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A conserved motif in the linker domain of STAT1 transcription factor is required for both recognition and release from high-affinity DNA-binding sites.
Bettina Hüntelmann, Julia Staab, Christoph Herrmann-Lingen, Thomas Meyer

PLoS One· January 2015

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DNA binding reduces the dissociation rate of STAT1 dimers and impairs the interdimeric exchange of protomers.
Theresa Riebeling, Julia Staab, Christoph Herrmann-Lingen, Thomas Meyer

BMC Biochem· December 2014

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CDK8 as the STAT1 serine 727 kinase?
Julia Staab, Christoph Herrmann-Lingen, Thomas Meyer

JAKSTAT· July 2013

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A rapid conformational rearrangement of STAT1 dimers is required for termination rather than for amplification of interferon-γ signaling.
Julia Staab, Christoph Herrmann-Lingen, Thomas Meyer

JAKSTAT· January 2013

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