Publications by authors named "Kavi Mehta"

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Viruses and the host replisome: discovering oncogenic mechanisms of small DNA tumor viruses.
Christopher D Collins, Matthew Stefely, Kavi Prem Milan Mehta, Megan E Spurgeon

J Virol· January 2026

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Ubiquitin-specific protease 15 interacts directly with the HSV-1 alkaline nuclease and facilitates viral recombination and replication fork stability.
Yee Vue, Patrick J Mullon, Alexander Leehangin, Emiliano Maldonado-Luevano, Tyler D Hasselmann, Kavi P M Mehta

J Virol· September 2025

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Adeno-Associated Virus 2 (AAV2) - Induced RPA exhaustion generates cellular DNA damage and restricts viral gene expression.
Monnette F Summers, MegAnn K Haubold, Marcel Morgenstern, Phoenix Shepherd, Clairine I S Larsen, Kavi P M Mehta

PLoS Pathog· August 2025

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SLX4IP acts in parallel to FANCM to limit BLM-dependent replication stress at ALT telomeres.
Jessica Spindler, Francesca Pandolfo, Anna Eva Koch, Priscilla Piccirillo, Johanna Bihler, Kavi P M Mehta

bioRxiv· May 2025

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The herpes simplex virus alkaline nuclease is required to maintain replication fork progression.
Patrick J Mullon, Emiliano Maldonado-Luevano, Kavi P M Mehta, Kareem N Mohni

J Virol· December 2024

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Contributing factors to the oxidation-induced mutational landscape in human cells.
Cameron Cordero, Kavi P M Mehta, Tyler M Weaver, Justin A Ling, Bret D Freudenthal

Nat Commun· December 2024

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Novel Gammapapillomavirus type in the nasal cavity of a wild red colobus (Piliocolobus tephrosceles).
Taylor E Weary, Kavi P M Mehta, Tony L Goldberg

Access Microbiol· August 2024

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Obesity Induces DNA Damage in Mammary Epithelial Cells Exacerbated by Acrylamide Treatment through CYP2E1-Mediated Oxidative Stress.
Brenna Walton, Noah Kaplan, Brooke Hrdlicka, Kavi Mehta, Lisa M Arendt

Toxics· July 2024

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The Causes and Consequences of DNA Damage and Chromosomal Instability Induced by Human Papillomavirus.
Kathryn M Jones, Ava Bryan, Emily McCunn, Pate E Lantz, Hunter Blalock, Kavi Mehta

Cancers (Basel)· April 2024

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Self-reversal facilitates the resolution of HMCES DNA-protein crosslinks in cells.
Jorge Rua-Fernandez, Courtney A Lovejoy, Kavi P M Mehta, Katherine A Paulin, Yasmine T Toudji

Cell Rep· November 2023

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Integrator facilitates RNAPII removal to prevent transcription-replication collisions and genome instability.
Rahul Bhowmick, Kavi P M Mehta, Mads Lerdrup, David Cortez

Mol Cell· July 2023

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Self-reversal facilitates the resolution of HMCES-DNA protein crosslinks in cells.
Jorge Rua-Fernandez, Courtney A Lovejoy, Kavi P M Mehta, Katherine A Paulin, Yasmine T Toudji

bioRxiv· June 2023

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Topoisomerase II poisons inhibit vertebrate DNA replication through distinct mechanisms.
Sabrina X Van Ravenstein, Kavi P Mehta, Tamar Kavlashvili, Jo Ann W Byl, Runxiang Zhao

EMBO J· June 2022

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CHK1 phosphorylates PRIMPOL to promote replication stress tolerance.
Kavi P M Mehta, Vaughn Thada, Runxiang Zhao, Archana Krishnamoorthy, Micheal Leser

Sci Adv· April 2022

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High-Risk Human Papillomaviruses and DNA Repair.
Kavi Mehta, Laimonis Laimins

Recent Results Cancer Res· January 2021

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HMCES Maintains Replication Fork Progression and Prevents Double-Strand Breaks in Response to APOBEC Deamination and Abasic Site Formation.
Kavi P M Mehta, Courtney A Lovejoy, Runxiang Zhao, Darren R Heintzman, David Cortez

Cell Rep· June 2020

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Vip1 is a kinase and pyrophosphatase switch that regulates inositol diphosphate signaling.
D Eric Dollins, Wenli Bai, Peter C Fridy, James C Otto, Julie L Neubauer, Kavi P M Mehta

Proc Natl Acad Sci U S A· April 2020

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HMCES Maintains Genome Integrity by Shielding Abasic Sites in Single-Strand DNA.
Kareem N Mohni, Sarah R Wessel, Runxiang Zhao, Andrea C Wojciechowski, Jessica W Luzwick, Kavi P M Mehta

Cell· January 2019

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Human Papillomaviruses Preferentially Recruit DNA Repair Factors to Viral Genomes for Rapid Repair and Amplification.
Kavi Mehta, Laimonis Laimins

mBio· February 2018

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Human papillomaviruses activate and recruit SMC1 cohesin proteins for the differentiation-dependent life cycle through association with CTCF insulators.
Kavi Mehta, Vignesh Gunasekharan, Ayano Satsuka, Laimonis A Laimins

PLoS Pathog· April 2015

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p38MAPK and MK2 pathways are important for the differentiation-dependent human papillomavirus life cycle.
Ayano Satsuka, Kavi Mehta, Laimonis Laimins

J Virol· February 2015

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Human papillomavirus E5 oncoproteins bind the A4 endoplasmic reticulum protein to regulate proliferative ability upon differentiation.
Katarina Kotnik Halavaty, Jennifer Regan, Kavi Mehta, Laimonis Laimins

Virology· March 2014

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Human papillomaviruses recruit cellular DNA repair and homologous recombination factors to viral replication centers.
Kenric A Gillespie, Kavi P Mehta, Laimonis A Laimins, Cary A Moody

J Virol· September 2012

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The E7 open reading frame acts in cis and in trans to mediate differentiation-dependent activities in the human papillomavirus type 16 life cycle.
Jason M Bodily, Kavi P M Mehta, Linda Cruz, Craig Meyers, Laimonis A Laimins

J Virol· September 2011

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Suppression of STAT-1 expression by human papillomaviruses is necessary for differentiation-dependent genome amplification and plasmid maintenance.
Shiyuan Hong, Kavi P Mehta, Laimonis A Laimins

J Virol· September 2011

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