Publications by authors named "Konrad A Bode"

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Interactions between the gut microbiome, associated metabolites and the manifestation and progression of heart failure with preserved ejection fraction in ZSF1 rats.
Salmina J Guivala, Konrad A Bode, Jürgen G Okun, Ece Kartal, Edzard Schwedhelm

Cardiovasc Diabetol· August 2024

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Gα modulates the energy metabolism of osteoclasts.
Sushmita Chakraborty, Bianca Handrick, Dayoung Yu, Konrad A Bode, Anna Hafner

Front Cell Infect Microbiol· January 2023

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Hsa-miR-99b/let-7e/miR-125a Cluster Regulates Pathogen Recognition Receptor-Stimulated Suppressive Antigen-Presenting Cells.
Dagmar Hildebrand, Mariel-Esther Eberle, Sabine Marie Wölfle, Franziska Egler, Delal Sahin, Konrad A Bode

Front Immunol· August 2019

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Human monocytes downregulate innate response receptors following exposure to the microbial metabolite n-butyrate.
Felix Lasitschka, Thomas Giese, Marco Paparella, Stefan R Kurzhals, Guido Wabnitz, Konrad A Bode

Immun Inflamm Dis· December 2017

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IL-1β As Mediator of Resolution That Reprograms Human Peripheral Monocytes toward a Suppressive Phenotype.
Katharina Giesbrecht, Mariel-Esther Eberle, Sabine J Wölfle, Delal Sahin, Aline Sähr, Konrad A Bode

Front Immunol· August 2017

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Evaluation of antibiotic resistance to orally administrable antibiotics in staphylococcal bone and joint infections in one of the largest university hospitals in Germany: is there a role for fusidic acid?
Sabrina Klein, Dennis Nurjadi, Tatjana Eigenbrod, Konrad A Bode

Int J Antimicrob Agents· February 2016

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Inhibition of Histone Deacetylases Permits Lipopolysaccharide-Mediated Secretion of Bioactive IL-1β via a Caspase-1-Independent Mechanism.
Dominik Stammler, Tatjana Eigenbrod, Sarah Menz, Julia S Frick, Matthew J Sweet, Konrad A Bode

J Immunol· December 2015

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Granzyme A produces bioactive IL-1β through a nonapoptotic inflammasome-independent pathway.
Dagmar Hildebrand, Konrad A Bode, David Rieß, Daniela Cerny, Anna Waldhuber

Cell Rep· November 2014

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Risk factors and outcome in patients with primary sclerosing cholangitis with persistent biliary candidiasis.
Christian Rupp, Konrad Alexander Bode, Fadi Chahoud, Andreas Wannhoff, Kilian Friedrich

BMC Infect Dis· October 2014

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IRAK4 turns IL-10+ phospho-FOXO+ monocytes into pro-inflammatory cells by suppression of protein kinase B.
Benjamin Over, Saskia Ziegler, Sandra Foermer, Alexander N R Weber, Konrad A Bode

Eur J Immunol· June 2013

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Early inhibition of IL-1β expression by IFN-γ is mediated by impaired binding of NF-κB to the IL-1β promoter but is independent of nitric oxide.
Tatjana Eigenbrod, Konrad A Bode, Alexander H Dalpke

J Immunol· June 2013

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Integration of matrix-assisted laser desorption/ionization time-of-flight mass spectrometry in blood culture diagnostics: a fast and effective approach.
Sabrina Klein, Stefan Zimmermann, Christine Köhler, Alexander Mischnik, Werner Alle, Konrad A Bode

J Med Microbiol· March 2012

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Distinct functions of the mitogen-activated protein kinase-activated protein (MAPKAP) kinases MK2 and MK3: MK2 mediates lipopolysaccharide-induced signal transducers and activators of transcription 3 (STAT3) activation by preventing negative regulatory effects of MK3.
Christian Ehlting, Natalia Ronkina, Oliver Böhmer, Ute Albrecht, Konrad A Bode, Johannes G Bode

J Biol Chem· July 2011

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Bronchial epithelial cell-derived prostaglandin E2 dampens the reactivity of dendritic cells.
Lotte M Schmidt, Maria G Belvisi, Konrad A Bode, Judith Bauer, Claudia Schmidt

J Immunol· February 2011

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HDAC inhibitors block innate immunity.
Konrad A Bode, Alexander H Dalpke

Blood· January 2011

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Kinetic of RelA activation controls magnitude of TLR-mediated IL-12p40 induction.
Konrad A Bode, Frank Schmitz, Leonardo Vargas, Klaus Heeg, Alexander H Dalpke

J Immunol· February 2009

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Histone deacetylase inhibitors decrease Toll-like receptor-mediated activation of proinflammatory gene expression by impairing transcription factor recruitment.
Konrad A Bode, Kate Schroder, David A Hume, Timothy Ravasi, Klaus Heeg

Immunology· December 2007

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Differential effects of CpG DNA on IFN-beta induction and STAT1 activation in murine macrophages versus dendritic cells: alternatively activated STAT1 negatively regulates TLR signaling in macrophages.
Kate Schroder, Martina Spille, Andreas Pilz, Jane Lattin, Konrad A Bode

J Immunol· September 2007

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Inhibition of transport across the hepatocyte canalicular membrane by the antibiotic fusidate.
Konrad A Bode, Markus G Donner, Inka Leier, Dietrich Keppler

Biochem Pharmacol· July 2002

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