Publications by authors named "Louise Appleton"

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Comparison of Cellular Responses to TGF-β1 and BMP-2 Between Healthy and Torn Tendons.
Wataru Morita, Sarah J B Snelling, Kim Wheway, Bridget Watkins, Louise Appleton

Am J Sports Med· June 2021

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Multi-omic single cell analysis resolves novel stromal cell populations in healthy and diseased human tendon.
Adrian R Kendal, Thomas Layton, Hussein Al-Mossawi, Louise Appleton, Stephanie Dakin

Sci Rep· September 2020

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ERK1/2 drives IL-1β-induced expression of TGF-β1 and BMP-2 in torn tendons.
Wataru Morita, Sarah J B Snelling, Kim Wheway, Bridget Watkins, Louise Appleton

Sci Rep· December 2019

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Proresolving Mediators LXB4 and RvE1 Regulate Inflammation in Stromal Cells from Patients with Shoulder Tendon Tears.
Stephanie G Dakin, Romain A Colas, Kim Wheway, Bridget Watkins, Louise Appleton

Am J Pathol· November 2019

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15-Epi-LXA and MaR1 counter inflammation in stromal cells from patients with Achilles tendinopathy and rupture.
Stephanie G Dakin, Romain A Colas, Julia Newton, Stephen Gwilym, Natasha Jones, Louise Appleton

FASEB J· July 2019

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The severity of ankylosing spondylitis and responses to anti-tumour necrosis factor biologics are not influenced by the tumour necrosis factor receptor polymorphism incriminated in multiple sclerosis.
Laura Watts, Tugce Karaderi, Amity Roberts, Louise Appleton, Tom Wordsworth

Genes Immun· February 2019

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Chronic inflammation is a feature of Achilles tendinopathy and rupture.
Stephanie Georgina Dakin, Julia Newton, Fernando O Martinez, Robert Hedley, Stephen Gwilym, Louise Appleton

Br J Sports Med· March 2018

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ERAP1 association with ankylosing spondylitis is attributable to common genotypes rather than rare haplotype combinations.
Amity R Roberts, Louise H Appleton, Adrian Cortes, Matteo Vecellio, Jonathan Lau

Proc Natl Acad Sci U S A· January 2017

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Mutations in Known Monogenic High Bone Mass Loci Only Explain a Small Proportion of High Bone Mass Cases.
Celia L Gregson, Lawrie Wheeler, Sarah A Hardcastle, Louise H Appleton, Kathryn A Addison

J Bone Miner Res· March 2016

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Major histocompatibility complex associations of ankylosing spondylitis are complex and involve further epistasis with ERAP1.
Adrian Cortes, Sara L Pulit, Paul J Leo, Jenny J Pointon, Philip C Robinson, Louise Appleton

Nat Commun· May 2015

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Identification of multiple risk variants for ankylosing spondylitis through high-density genotyping of immune-related loci.

Nat Genet· July 2013

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A common functional variant of endoplasmic reticulum aminopeptidase 2 (ERAP2) that reduces major histocompatibility complex class I expression is not associated with ankylosing spondylitis.
David Harvey, Jennifer J Pointon, Tugce Karaderi, Louise H Appleton, Claire Farrar

Rheumatology (Oxford)· September 2011

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Interaction between ERAP1 and HLA-B27 in ankylosing spondylitis implicates peptide handling in the mechanism for HLA-B27 in disease susceptibility.
David M Evans, Chris C A Spencer, Jennifer J Pointon, Zhan Su, David Harvey, Louise Appleton

Nat Genet· July 2011

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The chromosome 16q region associated with ankylosing spondylitis includes the candidate gene tumour necrosis factor receptor type 1-associated death domain (TRADD).
Jennifer J Pointon, David Harvey, Tugce Karaderi, Louise H Appleton, Claire Farrar

Ann Rheum Dis· June 2010

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Genome-wide association study of ankylosing spondylitis identifies non-MHC susceptibility loci.

Nat Genet· February 2010

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Investigating the genetic association between ERAP1 and ankylosing spondylitis.
David Harvey, Jennifer J Pointon, David M Evans, Tugce Karaderi, Claire Farrar, Louise H Appleton

Hum Mol Genet· November 2009

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Association between the interleukin 23 receptor and ankylosing spondylitis is confirmed by a new UK case-control study and meta-analysis of published series.
Tugce Karaderi, David Harvey, Claire Farrar, Louise H Appleton, Millicent A Stone

Rheumatology (Oxford)· April 2009

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