Publications by authors named "Marc Ittah"

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Induction of B cell-activating factor by viral infection is a general phenomenon, but the types of viruses and mechanisms depend on cell type.
Marc Ittah, Corinne Miceli-Richard, Pierre Lebon, Coralie Pallier, Christine Lepajolec

J Innate Immun· June 2011

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Functional study of TNF-α promoter polymorphisms: literature review and meta-analysis.
Arsene Mekinian, Ryad Tamouza, Stephan Pavy, Nicolas Gestermann, Marc Ittah

Eur Cytokine Netw· June 2011

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Effect of methotrexate and anti-TNF on Epstein-Barr virus T-cell response and viral load in patients with rheumatoid arthritis or spondylarthropathies.
Corinne Miceli-Richard, Nicolas Gestermann, Corinne Amiel, Jérémie Sellam, Marc Ittah

Arthritis Res Ther· March 2010

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Increased levels of circulating microparticles in primary Sjögren's syndrome, systemic lupus erythematosus and rheumatoid arthritis and relation with disease activity.
Jérémie Sellam, Valérie Proulle, Astrid Jüngel, Marc Ittah, Corinne Miceli Richard

Arthritis Res Ther· February 2010

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The CGGGG insertion/deletion polymorphism of the IRF5 promoter is a strong risk factor for primary Sjögren's syndrome.
Corinne Miceli-Richard, Nicolas Gestermann, Marc Ittah, Emmanuelle Comets, Pascale Loiseau

Arthritis Rheum· July 2009

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B-cell-activating factor expressions in salivary epithelial cells after dsRNA virus infection depends on RNA-activated protein kinase activation.
Marc Ittah, Corinne Miceli-Richard, Jacques-Eric Gottenberg, Jérémie Sellam, Christine Lepajolec

Eur J Immunol· May 2009

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Viruses induce high expression of BAFF by salivary gland epithelial cells through TLR- and type-I IFN-dependent and -independent pathways.
Marc Ittah, Corinne Miceli-Richard, Jacques-Eric Gottenberg, Jérémie Sellam, Pierre Eid

Eur J Immunol· April 2008

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Decreased B cell activating factor receptor expression on peripheral lymphocytes associated with increased disease activity in primary Sjögren's syndrome and systemic lupus erythematosus.
Jérémie Sellam, Corinne Miceli-Richard, Jacques-Eric Gottenberg, Marc Ittah, Frédéric Lavie

Ann Rheum Dis· June 2007

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Increase of B cell-activating factor of the TNF family (BAFF) after rituximab treatment: insights into a new regulating system of BAFF production.
Frédéric Lavie, Corinne Miceli-Richard, Marc Ittah, Jérémie Sellam, Jacques-Eric Gottenberg

Ann Rheum Dis· May 2007

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Tolerance and efficacy of rituximab and changes in serum B cell biomarkers in patients with systemic complications of primary Sjögren's syndrome.
Raphaèle Seror, Christelle Sordet, Loic Guillevin, Eric Hachulla, Charles Masson, Marc Ittah

Ann Rheum Dis· March 2007

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Late-onset neutropenia following rituximab results from a hematopoietic lineage competition due to an excessive BAFF-induced B-cell recovery.
Benjamin Terrier, Marc Ittah, Léa Tourneur, Fawzia Louache, Vassili Soumelis

Haematologica· February 2007

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B cell-activating factor of the tumor necrosis factor family (BAFF) is expressed under stimulation by interferon in salivary gland epithelial cells in primary Sjögren's syndrome.
Marc Ittah, Corinne Miceli-Richard, Jacques- Eric Gottenberg, Frédéric Lavie, Thierry Lazure

Arthritis Res Ther· August 2006

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No evidence for an association between the -871 T/C promoter polymorphism in the B-cell-activating factor gene and primary Sjögren's syndrome.
Jacques-Eric Gottenberg, Jérémie Sellam, Marc Ittah, Frédéric Lavie, Alexis Proust

Arthritis Res Ther· July 2006

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Failure to confirm coxsackievirus infection in primary Sjögren's syndrome.
Jacques-Eric Gottenberg, Coralie Pallier, Marc Ittah, Frédéric Lavie, Corinne Miceli-Richard

Arthritis Rheum· June 2006

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Animal models of HLA-B27-associated diseases: new outcomes.
Cécile Hacquard-Bouder, Marc Ittah, Maxime Breban

Joint Bone Spine· March 2006

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Activation of IFN pathways and plasmacytoid dendritic cell recruitment in target organs of primary Sjögren's syndrome.
Jacques-Eric Gottenberg, Nicolas Cagnard, Carlo Lucchesi, Franck Letourneur, Sylvie Mistou, Marc Ittah

Proc Natl Acad Sci U S A· February 2006

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Defective costimulatory function is a striking feature of antigen-presenting cells in an HLA-B27-transgenic rat model of spondylarthropathy.
Cécile Hacquard-Bouder, Géraldine Falgarone, Antoine Bosquet, Faïza Smaoui, Dominique Monnet, Marc Ittah

Arthritis Rheum· May 2004

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