Publications by authors named "Masami Ojima"

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Landscape of driver mutations and their clinical effects on Down syndrome-related myeloid neoplasms.
Tomohiko Sato, Kenichi Yoshida, Tsutomu Toki, Rika Kanezaki, Kiminori Terui, Masami Ojima

Blood· June 2024

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Exploring Large MAF Transcription Factors: Functions, Pathology, and Mouse Models with Point Mutations.
Mitsunori Fujino, Masami Ojima, Satoru Takahashi

Genes (Basel)· September 2023

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Generation and mutational analysis of a transgenic murine model of the human MAF mutation.
Mitsunori Fujino, Masami Ojima, Shun Ishibashi, Seiya Mizuno, Satoru Takahashi

Am J Med Genet A· July 2023

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Transcription factor c-Maf deletion improves streptozotocin-induced diabetic nephropathy by directly regulating Sglt2 and Glut2.
Mitsunori Fujino, Naoki Morito, Takuto Hayashi, Masami Ojima, Shun Ishibashi

JCI Insight· March 2023

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MafB Maintains -Cell Identity under MafA-Deficient Conditions.
Zhaobin Deng, Akihiro Kuno, Masami Ojima, Satoru Takahashi

Mol Cell Biol· August 2022

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An Inducible Diabetes Mellitus Murine Model Based on MafB Conditional Knockout under MafA-Deficient Condition.
Zhaobin Deng, Yuka Matsumoto, Akihiro Kuno, Masami Ojima, Gulibaikelamu Xiafukaiti

Int J Mol Sci· August 2020

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Uncovering the role of MAFB in glucagon production and secretion in pancreatic α-cells using a new α-cell-specific Mafb conditional knockout mouse model.
Yu-Hsin Chang, Megumi C Katoh, Ahmed M Abdellatif, Guli Xiafukaiti, Abdelaziz Elzeftawy, Masami Ojima

Exp Anim· April 2020

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c-MAF deletion in adult C57BL/6J mice induces cataract formation and abnormal differentiation of lens fiber cells.
Mitsunori Fujino, Asuka Tagami, Masami Ojima, Seiya Mizuno, Ahmed M Abdellatif

Exp Anim· April 2020

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Phenotypic analysis of mice carrying human-type MAFB p.Leu239Pro mutation.
Maho Kanai, Hyojung Jeon, Masami Ojima, Teppei Nishino, Toshiaki Usui

Biochem Biophys Res Commun· March 2020

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Neuron-specific Mafb knockout causes growth retardation accompanied by an impaired growth hormone/insulin-like growth factor I axis.
Shayida Maimaiti, Ryusuke Koshida, Masami Ojima, Kaushalya Kulathunga, Hisashi Oishi

Exp Anim· November 2019

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Transcription factor MafB may play an important role in secondary hyperparathyroidism.
Naoki Morito, Keigyou Yoh, Toshiaki Usui, Hisashi Oishi, Masami Ojima

Kidney Int· January 2018

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Th2-biased GATA-3 transgenic mice developed severe experimental peritoneal fibrosis compared with Th1-biased T-bet and Th17-biased RORγt transgenic mice.
Keigyou Yoh, Masami Ojima, Satoru Takahashi

Exp Anim· June 2016

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Overexpression of Mafb in podocytes protects against diabetic nephropathy.
Naoki Morito, Keigyou Yoh, Masami Ojima, Midori Okamura, Megumi Nakamura

J Am Soc Nephrol· November 2014

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Overexpression of GATA-3 in T cells accelerates dextran sulfate sodium-induced colitis.
Midori Okamura, Keigyou Yoh, Masami Ojima, Naoki Morito, Satoru Takahashi

Exp Anim· October 2014

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Overexpression of RORγt under control of the CD2 promoter induces polyclonal plasmacytosis and autoantibody production in transgenic mice.
Keigyou Yoh, Naoki Morito, Masami Ojima, Kazuko Shibuya, Yumi Yamashita

Eur J Immunol· August 2012

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A novel diabetes mellitus mouse model, MAFA-deficient and beta cell-specific MAFK-overexpressing hybrid transgenic mice, developed severe diabetic nephropathy and improved with TCV-116 (candesartan cilexetil) treatment.
Akiko Fujita, Keigyou Yoh, Homare Shimohata, Naoki Morito, Masami Ojima

Exp Anim· May 2012

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MafA-deficient and beta cell-specific MafK-overexpressing hybrid transgenic mice develop human-like severe diabetic nephropathy.
Homare Shimohata, Keigyou Yoh, Akiko Fujita, Naoki Morito, Masami Ojima

Biochem Biophys Res Commun· November 2009

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Hyperglycemia induces oxidative and nitrosative stress and increases renal functional impairment in Nrf2-deficient mice.
Keigyou Yoh, Aki Hirayama, Kazusa Ishizaki, Akiko Yamada, Masayoshi Takeuchi, Masami Ojima

Genes Cells· November 2008

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