Publications by authors named "Matthew J Hart"

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Targeting DDR2 for Treating Pancreatic Cancer.
Chris T P Do, Prabhakar Pitta Venkata, Jack Y Prochnau, Deepika Singh, Santosh Timilsina, Matthew J Hart

Mol Cancer Ther· October 2025

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Hypersensitive intercellular responses of endometrial stromal cells drive invasion in endometriosis.
Chun-Wei Chen, Jeffery B Chavez, Ritikaa Kumar, Virginia Arlene Go, Ahvani Pant, Matthew J Hart

Elife· December 2024

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Soluble pathogenic tau enters brain vascular endothelial cells and drives cellular senescence and brain microvascular dysfunction in a mouse model of tauopathy.
Stacy A Hussong, Andy Q Banh, Candice E Van Skike, Angela O Dorigatti, Stephen F Hernandez, Matthew J Hart

Nat Commun· April 2023

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mTOR drives cerebrovascular, synaptic, and cognitive dysfunction in normative aging.
Candice E Van Skike, Ai-Ling Lin, Raquel Roberts Burbank, Jonathan J Halloran, Stephen F Hernandez, Matthew J Hart

Aging Cell· January 2020

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Corrigendum to "Synthesis and SAR of novel capsazepine analogs with significant anti-cancer effects in multiple cancer types" [Bioorg. Med. Chem. 27 (1) (2019) 208-215].
Jorge De La Chapa, Matthew Valdez, Francisco Ruiz, Keith Gonzales, Wes Mitchell, Matthew J Hart

Bioorg Med Chem· June 2019

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The novel capsazepine analog, CIDD-99, significantly inhibits oral squamous cell carcinoma in vivo through a TRPV1-independent induction of ER stress, mitochondrial dysfunction, and apoptosis.
Jorge J De La Chapa, Prajjal K Singha, Kristen K Self, McKay L Sallaway, Stanton F McHardy, Matthew J Hart

J Oral Pathol Med· May 2019

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Luteolin inhibits Musashi1 binding to RNA and disrupts cancer phenotypes in glioblastoma cells.
Caihong Yi, Guiming Li, Dmitri N Ivanov, Zhonghua Wang, Mitzli X Velasco, Matthew J Hart

RNA Biol· January 2019

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An engineered transforming growth factor β (TGF-β) monomer that functions as a dominant negative to block TGF-β signaling.
Sun Kyung Kim, Lindsey Barron, Cynthia S Hinck, Elyse M Petrunak, Kristin E Cano, P John Hart, Matthew J Hart

J Biol Chem· April 2017

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Vascular mTOR-dependent mechanisms linking the control of aging to Alzheimer's disease.
Veronica Galvan, Matthew J Hart

Biochim Biophys Acta· May 2016

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Rapamycin improves motor function, reduces 4-hydroxynonenal adducted protein in brain, and attenuates synaptic injury in a mouse model of synucleinopathy.
Xiang Bai, Margaret Chia-Ying Wey, Elizabeth Fernandez, Matthew J Hart, Jonathan Gelfond

Pathobiol Aging Age Relat Dis· August 2015

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Paradoxical effect of TrkA inhibition in Alzheimer's disease models.
Qiang Zhang, Olivier Descamps, Matthew J Hart, Karen S Poksay, Patricia Spilman

J Alzheimers Dis· December 2014

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Chronic rapamycin restores brain vascular integrity and function through NO synthase activation and improves memory in symptomatic mice modeling Alzheimer's disease.
Ai-Ling Lin, Wei Zheng, Jonathan J Halloran, Raquel R Burbank, Stacy A Hussong, Matthew J Hart

J Cereb Blood Flow Metab· September 2013

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Over-expression of heat shock factor 1 phenocopies the effect of chronic inhibition of TOR by rapamycin and is sufficient to ameliorate Alzheimer's-like deficits in mice modeling the disease.
Anson Pierce, Natalia Podlutskaya, Jonathan J Halloran, Stacy A Hussong, Pei-Yi Lin, Matthew J Hart

J Neurochem· March 2013

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The role of MAP4K3 in lifespan regulation of Caenorhabditis elegans.
Maruf H Khan, Matthew J Hart, Shane L Rea

Biochem Biophys Res Commun· August 2012

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Development of a high-throughput screen targeting caspase-8-mediated cleavage of the amyloid precursor protein.
Matthew J Hart, Marcie Glicksman, Min Liu, Mohit K Sharma, Greg Cuny

Anal Biochem· February 2012

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Endogenously EGFP-Labeled Mouse Embryonic Stem Cells.
Junli Zhang, Rammohan V Rao, Patricia Spilman, Julie Mangada, Lin Xie, Matthew J Hart

Aging Dis· February 2011

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Inhibition of mTOR by rapamycin abolishes cognitive deficits and reduces amyloid-beta levels in a mouse model of Alzheimer's disease.
Patricia Spilman, Natalia Podlutskaya, Matthew J Hart, Jayanta Debnath, Olivia Gorostiza

PLoS One· April 2010

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Platelet endothelial aggregation receptor 1 (PEAR1), a novel epidermal growth factor repeat-containing transmembrane receptor, participates in platelet contact-induced activation.
Nisha Nanda, Ming Bao, Hanna Lin, Karl Clauser, Laszlo Komuves, Matthew J Hart

J Biol Chem· July 2005

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