Publications by authors named "Miha Pakusch"

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A minority of proliferating human CD4 T cells in antigen-driven proliferation assays are antigen specific.
Pushpak Bhattacharjee, Miha Pakusch, Matthew Lacorcia, Chris Y Chiu, Xin Liu

Front Immunol· November 2024

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Proinsulin C-peptide is a major source of HLA-DQ8 restricted hybrid insulin peptides recognized by human islet-infiltrating CD4 T cells.
Pushpak Bhattacharjee, Miha Pakusch, Matthew Lacorcia, Eleonora Tresoldi, Alan F Rubin

PNAS Nexus· November 2024

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Glutamine deamidation does not increase the immunogenicity of C-peptide in people with type 1 diabetes.
Abby Foster, Pushpak Bhattacharjee, Eleonora Tresoldi, Miha Pakusch, Fergus J Cameron

J Transl Autoimmun· December 2022

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BAF complex-mediated chromatin relaxation is required for establishment of X chromosome inactivation.
Andrew Keniry, Natasha Jansz, Linden J Gearing, Iromi Wanigasuriya, Joseph Chen, Miha Pakusch

Nat Commun· March 2022

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Proinsulin C-peptide is an autoantigen in people with type 1 diabetes.
Michelle So, Colleen M Elso, Eleonora Tresoldi, Miha Pakusch, Vimukthi Pathiraja

Proc Natl Acad Sci U S A· October 2018

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Smchd1 haploinsufficiency exacerbates the phenotype of a transgenic FSHD1 mouse model.
Jessica C de Greef, Yvonne D Krom, Bianca den Hamer, Lauren Snider, Yosuke Hiramuki, Miha Pakusch

Hum Mol Genet· February 2018

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Setdb1-mediated H3K9 methylation is enriched on the inactive X and plays a role in its epigenetic silencing.
Andrew Keniry, Linden J Gearing, Natasha Jansz, Joy Liu, Aliaksei Z Holik, Miha Pakusch

Epigenetics Chromatin· May 2016

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Smchd1 regulates a subset of autosomal genes subject to monoallelic expression in addition to being critical for X inactivation.
Arne W Mould, Zhenyi Pang, Miha Pakusch, Ian D Tonks, Mitchell Stark

Epigenetics Chromatin· July 2013

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Epigenetic regulator Smchd1 functions as a tumor suppressor.
Huei San Leong, Kelan Chen, Yifang Hu, Stanley Lee, Jason Corbin, Miha Pakusch

Cancer Res· March 2013

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Opposing roles of polycomb repressive complexes in hematopoietic stem and progenitor cells.
Ian J Majewski, Matthew E Ritchie, Belinda Phipson, Jason Corbin, Miha Pakusch

Blood· August 2010

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Cell death provoked by loss of interleukin-3 signaling is independent of Bad, Bim, and PI3 kinase, but depends in part on Puma.
Paul G Ekert, Anissa M Jabbour, Anand Manoharan, Jacki E Heraud, Jai Yu, Miha Pakusch

Blood· September 2006

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Determination of cell survival by RING-mediated regulation of inhibitor of apoptosis (IAP) protein abundance.
John Silke, Tobias Kratina, Diep Chu, Paul G Ekert, Catherine L Day, Miha Pakusch

Proc Natl Acad Sci U S A· November 2005

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Unlike Diablo/smac, Grim promotes global ubiquitination and specific degradation of X chromosome-linked inhibitor of apoptosis (XIAP) and neither cause apoptosis.
John Silke, Tobias Kratina, Paul G Ekert, Miha Pakusch, David L Vaux

J Biol Chem· February 2004

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The anti-apoptotic activity of XIAP is retained upon mutation of both the caspase 3- and caspase 9-interacting sites.
John Silke, Christine J Hawkins, Paul G Ekert, Joanne Chew, Catherine L Day, Miha Pakusch

J Cell Biol· April 2002

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HtrA2 promotes cell death through its serine protease activity and its ability to antagonize inhibitor of apoptosis proteins.
Anne M Verhagen, John Silke, Paul G Ekert, Miha Pakusch, Hitto Kaufmann

J Biol Chem· January 2002

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