Publications by authors named "Nico Hartmann"

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Atrial fibrillation in end-stage heart failure: Cellular mechanisms behind CASTLE-HTx.
Maria Knierim, Nico Hartmann, Wiebke Maurer, Steffen Pabel, Simon Sedej

Eur J Heart Fail· September 2025

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Na1.8 as Proarrhythmic Target in a Ventricular Cardiac Stem Cell Model.
Nico Hartmann, Maria Knierim, Wiebke Maurer, Nataliya Dybkova, Florian Zeman

Int J Mol Sci· June 2024

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Molecular and Functional Relevance of Na1.8-Induced Atrial Arrhythmogenic Triggers in a Human Knock-Out Stem Cell Model.
Nico Hartmann, Maria Knierim, Wiebke Maurer, Nataliya Dybkova, Gerd Hasenfuß

Int J Mol Sci· June 2023

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Electrophysiological and calcium-handling development during long-term culture of human-induced pluripotent stem cell-derived cardiomyocytes.
Fitzwilliam Seibertz, Henry Sutanto, Rebekka Dülk, Julius Ryan D Pronto, Robin Springer, Nico Hartmann

Basic Res Cardiol· April 2023

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Different activation of MAPKs and Akt/GSK3β after preload vs. afterload elevation.
Nico Hartmann, Lena Preuß, Belal A Mohamed, Moritz Schnelle, Andre Renner

ESC Heart Fail· June 2022

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Generation of homozygous Na1.8 knock-out iPSC lines by CRISPR Cas9 genome editing to investigate a potential new antiarrhythmic strategy.
Wiebke Maurer, Nico Hartmann, Loukas Argyriou, Samuel Sossalla, Katrin Streckfuss-Bömeke

Stem Cell Res· April 2022

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Detrimental proarrhythmogenic interaction of Ca/calmodulin-dependent protein kinase II and Na1.8 in heart failure.
Philipp Bengel, Nataliya Dybkova, Petros Tirilomis, Shakil Ahmad, Nico Hartmann

Nat Commun· November 2021

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The functional consequences of sodium channel Na 1.8 in human left ventricular hypertrophy.
Shakil Ahmad, Petros Tirilomis, Steffen Pabel, Nataliya Dybkova, Nico Hartmann

ESC Heart Fail· February 2019

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Differential regulation of sodium channels as a novel proarrhythmic mechanism in the human failing heart.
Nataliya Dybkova, Shakil Ahmad, Steffen Pabel, Petros Tirilomis, Nico Hartmann

Cardiovasc Res· November 2018

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Sarcoplasmic reticulum calcium leak contributes to arrhythmia but not to heart failure progression.
Belal A Mohamed, Nico Hartmann, Petros Tirilomis, Karolina Sekeres, Wener Li

Sci Transl Med· September 2018

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Antiarrhythmic effects of dantrolene in human diseased cardiomyocytes.
Nico Hartmann, Steffen Pabel, Jonas Herting, Felix Schatter, André Renner

Heart Rhythm· March 2017

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Sex-dependent alterations of Ca2+ cycling in human cardiac hypertrophy and heart failure.
Thomas H Fischer, Jonas Herting, Jörg Eiringhaus, Steffen Pabel, Nico H Hartmann

Europace· September 2016

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Enhanced cardiac TBC1D10C expression lowers heart rate and enhances exercise capacity and survival.
Cornelia Volland, Sebastian Bremer, Kristian Hellenkamp, Nico Hartmann, Nataliya Dybkova

Sci Rep· September 2016

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Inhibition of CaMKII Attenuates Progressing Disruption of Ca(2+) Homeostasis Upon Left Ventricular Assist Device Implantation in Human Heart Failure.
Thomas H Fischer, Astrid Kleinwächter, Jonas Herting, Jörg Eiringhaus, Nico Hartmann

Artif Organs· August 2016

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The combined effects of ranolazine and dronedarone on human atrial and ventricular electrophysiology.
Nico Hartmann, Fleur E Mason, Inga Braun, Steffen Pabel, Niels Voigt

J Mol Cell Cardiol· May 2016

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Late INa increases diastolic SR-Ca2+-leak in atrial myocardium by activating PKA and CaMKII.
Thomas H Fischer, Jonas Herting, Fleur E Mason, Nico Hartmann, Saera Watanabe

Cardiovasc Res· July 2015

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Long term clinical outcome of dental implants placed in a patient with Singleton-Merten syndrome.
Reena Rodriguez, Nico Hartmann, Ludger Figgener, Johannes Kleinheinz, Dieter Weingart

J Prosthodont Res· July 2015

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Enhanced late INa induces proarrhythmogenic SR Ca leak in a CaMKII-dependent manner.
Can M Sag, Anika Mallwitz, Stefan Wagner, Nico Hartmann, Hanna Schotola

J Mol Cell Cardiol· November 2014

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Role of late sodium current as a potential arrhythmogenic mechanism in the progression of pressure-induced heart disease.
Karl Toischer, Nico Hartmann, Stefan Wagner, Thomas H Fischer, Jonas Herting

J Mol Cell Cardiol· August 2013

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Diastolic dysfunction and arrhythmias caused by overexpression of CaMKIIδ(C) can be reversed by inhibition of late Na(+) current.
Samuel Sossalla, Ulrike Maurer, Hanna Schotola, Nico Hartmann, Michael Didié

Basic Res Cardiol· March 2011

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