Publications by authors named "Sayoko Fujimura"

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Atypical heat shock transcription factor HSF5 is critical for male meiotic prophase under non-stress conditions.
Saori Yoshimura, Ryuki Shimada, Koji Kikuchi, Soichiro Kawagoe, Hironori Abe, Sayoko Fujimura

Nat Commun· April 2024

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STRA8-RB interaction is required for timely entry of meiosis in mouse female germ cells.
Ryuki Shimada, Yuzuru Kato, Naoki Takeda, Sayoko Fujimura, Kei-Ichiro Yasunaga

Nat Commun· October 2023

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Nutritional control of thyroid morphogenesis through gastrointestinal hormones.
Maki Takagishi, Binta Maria Aleogho, Masako Okumura, Kaori Ushida, Yuichiro Yamada, Sayoko Fujimura

Curr Biol· April 2022

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FBXO47 is essential for preventing the synaptonemal complex from premature disassembly in mouse male meiosis.
Nobuhiro Tanno, Kazumasa Takemoto, Yuki Takada-Horisawa, Ryuki Shimada, Sayoko Fujimura

iScience· April 2022

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Meiosis-specific ZFP541 repressor complex promotes developmental progression of meiotic prophase towards completion during mouse spermatogenesis.
Yuki Horisawa-Takada, Chisato Kodera, Kazumasa Takemoto, Akihiko Sakashita, Kenichi Horisawa, Sayoko Fujimura

Nat Commun· June 2021

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Meiotic cohesins mediate initial loading of HORMAD1 to the chromosomes and coordinate SC formation during meiotic prophase.
Yasuhiro Fujiwara, Yuki Horisawa-Takada, Erina Inoue, Naoki Tani, Hiroki Shibuya, Sayoko Fujimura

PLoS Genet· September 2020

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Phosphorylation of the Anaphase Promoting Complex activator FZR1/CDH1 is required for Meiosis II entry in mouse male germ cell.
Nobuhiro Tanno, Shinji Kuninaka, Sayoko Fujimura, Kazumasa Takemoto, Kaho Okamura

Sci Rep· June 2020

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Meiosis-Specific C19orf57/4930432K21Rik/BRME1 Modulates Localization of RAD51 and DMC1 to DSBs in Mouse Meiotic Recombination.
Kazumasa Takemoto, Naoki Tani, Yuki Takada-Horisawa, Sayoko Fujimura, Nobuhiro Tanno

Cell Rep· May 2020

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MEIOSIN Directs the Switch from Mitosis to Meiosis in Mammalian Germ Cells.
Kei-Ichiro Ishiguro, Kumi Matsuura, Naoki Tani, Naoki Takeda, Shingo Usuki, Sayoko Fujimura

Dev Cell· February 2020

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Non-muscle myosin II deletion in the developing kidney causes ureter-bladder misconnection and apical extrusion of the nephric duct lineage epithelia.
Fahim Haque, Yusuke Kaku, Sayoko Fujimura, Tomoko Ohmori, Robert S Adelstein

Dev Biol· July 2017

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Sall1 in renal stromal progenitors non-cell autonomously restricts the excessive expansion of nephron progenitors.
Tomoko Ohmori, Shunsuke Tanigawa, Yusuke Kaku, Sayoko Fujimura, Ryuichi Nishinakamura

Sci Rep· October 2015

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Nonmuscle Myosin II Regulates the Morphogenesis of Metanephric Mesenchyme-Derived Immature Nephrons.
Mariam C Recuenco, Tomoko Ohmori, Shunsuke Tanigawa, Atsuhiro Taguchi, Sayoko Fujimura

J Am Soc Nephrol· May 2015

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Islet1 deletion causes kidney agenesis and hydroureter resembling CAKUT.
Yusuke Kaku, Tomoko Ohmori, Kuniko Kudo, Sayoko Fujimura, Kentaro Suzuki

J Am Soc Nephrol· July 2013

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The phosphatase Dullard negatively regulates BMP signalling and is essential for nephron maintenance after birth.
Masaji Sakaguchi, Sazia Sharmin, Atsuhiro Taguchi, Tomoko Ohmori, Sayoko Fujimura

Nat Commun· June 2013

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Nephron progenitors in the metanephric mesenchyme.
Ryuichi Nishinakamura, Yukako Uchiyama, Masaji Sakaguchi, Sayoko Fujimura

Pediatr Nephrol· September 2011

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Notch2 activation in the embryonic kidney depletes nephron progenitors.
Sayoko Fujimura, Qing Jiang, Chiyoko Kobayashi, Ryuichi Nishinakamura

J Am Soc Nephrol· May 2010

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Kif26b, a kinesin family gene, regulates adhesion of the embryonic kidney mesenchyme.
Yukako Uchiyama, Masaji Sakaguchi, Takeshi Terabayashi, Toshiaki Inenaga, Shuji Inoue, Sayoko Fujimura

Proc Natl Acad Sci U S A· May 2010

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Overexpression of Sall1 in vivo leads to reduced body weight without affecting kidney development.
Qing Jiang, Sayoko Fujimura, Chiyoko Kobayashi, Ryuichi Nishinakamura

J Biochem· March 2010

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A mouse line expressing Sall1-driven inducible Cre recombinase in the kidney mesenchyme.
Shuji Inoue, Miki Inoue, Sayoko Fujimura, Ryuichi Nishinakamura

Genesis· March 2010

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Sall4 is essential for stabilization, but not for pluripotency, of embryonic stem cells by repressing aberrant trophectoderm gene expression.
Shunsuke Yuri, Sayoko Fujimura, Keisuke Nimura, Naoki Takeda, Yayoi Toyooka, Yu-Ichi Fujimura

Stem Cells· April 2009

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The murine homolog of SALL4, a causative gene in Okihiro syndrome, is essential for embryonic stem cell proliferation, and cooperates with Sall1 in anorectal, heart, brain and kidney development.
Masayo Sakaki-Yumoto, Chiyoko Kobayashi, Akira Sato, Sayoko Fujimura, Yuko Matsumoto

Development· August 2006

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