Publications by authors named "Thomas Hackenbeck"

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Timely Monitoring of SARS-CoV-2 RNA Fragments in Wastewater Shows the Emergence of JN.1 (BA.2.86.1.1, Clade 23I) in Berlin, Germany.
Alexander Bartel, José Horacio Grau, Julia Bitzegeio, Dirk Werber, Nico Linzner, Thomas Hackenbeck

Viruses· January 2024

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SARS-CoV-2 infection dynamics revealed by wastewater sequencing analysis and deconvolution.
Vic-Fabienne Schumann, Rafael Ricardo de Castro Cuadrat, Emanuel Wyler, Ricardo Wurmus, Aylina Deter, Thomas Hackenbeck

Sci Total Environ· December 2022

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Biallelic Expression of Mucin-1 in Autosomal Dominant Tubulointerstitial Kidney Disease: Implications for Nongenetic Disease Recognition.
Karl X Knaup, Thomas Hackenbeck, Bernt Popp, Johanna Stoeckert, Andrea Wenzel

J Am Soc Nephrol· September 2018

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Hypoxia inhibits nephrogenesis through paracrine Vegfa despite the ability to enhance tubulogenesis.
Gunnar Schley, Holger Scholz, Andre Kraus, Thomas Hackenbeck, Bernd Klanke

Kidney Int· December 2015

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Hypoxia regulates the sperm associated antigen 4 (SPAG4) via HIF, which is expressed in renal clear cell carcinoma and promotes migration and invasion in vitro.
Karl Xaver Knaup, Juliana Monti, Thomas Hackenbeck, Tilmann Jobst-Schwan, Bernd Klanke

Mol Carcinog· December 2014

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Renal fibrosis is the common feature of autosomal dominant tubulointerstitial kidney diseases caused by mutations in mucin 1 or uromodulin.
Arif B Ekici, Thomas Hackenbeck, Vincent Morinière, Andrea Pannes, Maike Buettner

Kidney Int· September 2014

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Renal uptake of the antiapoptotic protein survivin is mediated by megalin at the apical membrane of the proximal tubule.
Tilman Jobst-Schwan, Karl X Knaup, Rikke Nielsen, Thomas Hackenbeck, Maike Buettner-Herold

Am J Physiol Renal Physiol· September 2013

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Renal tubular HIF-2α expression requires VHL inactivation and causes fibrosis and cysts.
Ruth E Schietke, Thomas Hackenbeck, Maxine Tran, Regina Günther, Bernd Klanke

PLoS One· July 2012

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The GTPase RAB20 is a HIF target with mitochondrial localization mediating apoptosis in hypoxia.
Thomas Hackenbeck, Regina Huber, Ruth Schietke, Karl X Knaup, Juliana Monti

Biochim Biophys Acta· January 2011

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Hypoxia-inducible protein 2 is a novel lipid droplet protein and a specific target gene of hypoxia-inducible factor-1.
Tina Gimm, Melanie Wiese, Barbara Teschemacher, Anke Deggerich, Johannes Schödel, Thomas Hackenbeck

FASEB J· November 2010

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HIF-1 or HIF-2 induction is sufficient to achieve cell cycle arrest in NIH3T3 mouse fibroblasts independent from hypoxia.
Thomas Hackenbeck, Karl Xaver Knaup, Ruth Schietke, Johannes Schödel, Carsten Willam

Cell Cycle· May 2009

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The specific contribution of hypoxia-inducible factor-2alpha to hypoxic gene expression in vitro is limited and modulated by cell type-specific and exogenous factors.
Christina Warnecke, Alexander Weidemann, Melanie Volke, Ruth Schietke, Xiaoqing Wu, Thomas Hackenbeck

Exp Cell Res· June 2008

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The tumor gene survivin is highly expressed in adult renal tubular cells: implications for a pathophysiological role in the kidney.
Philipp Lechler, Xiaoqing Wu, Wanja Bernhardt, Valentina Campean, Susanne Gastiger, Thomas Hackenbeck

Am J Pathol· November 2007

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DNase I treatment of cDNA first strands prevents RT-PCR amplification of contaminating DNA sequences.
Aljoscha M Flohr, Thomas Hackenbeck, Claudia Schlueter, Piere Rogalla, Joern Bullerdiek

Biotechniques· November 2003

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