Publications by authors named "Trisha Finlay"

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Presence and activation of pro-inflammatory macrophages are associated with CRYAB expression in vitro and after peripheral nerve injury.
Erin-Mai F Lim, Vahid Hoghooghi, Kathleen M Hagen, Kunal Kapoor, Ariana Frederick, Trisha M Finlay

J Neuroinflammation· March 2021

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Cystatin C Plays a Sex-Dependent Detrimental Role in Experimental Autoimmune Encephalomyelitis.
Vahid Hoghooghi, Alexandra L Palmer, Ariana Frederick, Yulan Jiang, Jessica E Merkens, Trisha M Finlay

Cell Rep· October 2020

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Murine neutrophils treated with alphaB-crystallin reduce IL-12p40 production by dendritic cells.
Trisha M Finlay, Alexandra L Palmer, Shalina S Ousman

Immunology· September 2018

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Characterization of migration parameters on peripheral and central nervous system T cells following treatment of experimental allergic encephalomyelitis with CRYAB.
Adeola I Oyebamiji, Trisha M Finlay, Rachael M Hough, Vahid Hoghooghi, Erin-Mai F Lim

J Neuroimmunol· June 2013

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Neu1 sialidase and matrix metalloproteinase-9 cross-talk is essential for Toll-like receptor activation and cellular signaling.
Samar Abdulkhalek, Schammim Ray Amith, Susan L Franchuk, Preethi Jayanth, Merry Guo, Trisha Finlay

J Biol Chem· October 2011

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Detection of Neu1 sialidase activity in regulating Toll-like receptor activation.
Schammim R Amith, Preethi Jayanth, Trisha Finlay, Susan Franchuk, Alanna Gilmour

J Vis Exp· September 2010

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Thymoquinone-induced Neu4 sialidase activates NFκB in macrophage cells and pro-inflammatory cytokines in vivo.
Trisha M Finlay, Samar Abdulkhalek, Alanna Gilmour, Christina Guzzo, Preethi Jayanth

Glycoconj J· August 2010

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Thymoquinone from nutraceutical black cumin oil activates Neu4 sialidase in live macrophage, dendritic, and normal and type I sialidosis human fibroblast cells via GPCR Galphai proteins and matrix metalloproteinase-9.
Trisha M Finlay, Preethi Jayanth, Schammim Ray Amith, Alanna Gilmour, Christina Guzzo

Glycoconj J· April 2010

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Substrain differences reveal novel disease-modifying gene candidates that alter the clinical course of a rodent model of multiple sclerosis.
Leslie E Summers deLuca, Natalia B Pikor, Jennifer O'Leary, Georgina Galicia-Rosas, Lesley A Ward, Trisha M Finlay

J Immunol· March 2010

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Neu1 desialylation of sialyl alpha-2,3-linked beta-galactosyl residues of TOLL-like receptor 4 is essential for receptor activation and cellular signaling.
Schammim Ray Amith, Preethi Jayanth, Susan Franchuk, Trisha Finlay, Volkan Seyrantepe

Cell Signal· February 2010

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