@FatEmperor @LDLSkeptic @Drlipid @TrueHealthDiag @TuckerGoodrich @daveketo @siobhan_huggins That’s why the indices TG/HDL and TyG usually correlate much better with CVD risk than LDL-C. https://t.co/4QQuwVcziX https://t.co/o8nDuf8uGc
Pathophysiology of human visceral obesity: an update.
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Nov 2017 – Nov 2025Where it's discussed
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What people are saying
This collection of discussions demonstrates a strong interest in the biological complexities of visceral obesity, with references to the key contributions of Tchernof and Després, and mentions of measurement issues and hormonal influences. While some comments simply acknowledge the study’s significance, others delve into specific mechanisms, indicating a moderate level of engagement and perceived impact in ongoing health debates.
Most discussions reference the importance and validity of the research, with users acknowledging its foundational insights and impact on understanding obesity.
The posts show high interest, with users actively highlighting specific aspects like visceral fat's role and its measurement limitations.
Some shared personal insights and questions about mechanisms, indicating moderate engagement; however, many posts remain superficial acknowledgments.
The discussions suggest that the article influences ongoing conversations, especially regarding health implications and biological factors, although some comments are more casual or tangential.
Abstract in brief
- Visceral obesity involves the accumulation of intraabdominal fat that is linked to several metabolic problems like high triglycerides, increased free fatty acids, inflammation, insulin resistance, and unhealthy lipoprotein profiles, all contributing to increased cardiometabolic risk.
- Factors such as age, gender, genetics, and ethnicity influence how much visceral fat a person accumulates, with mechanisms like hormonal changes, cortisol production, endocannabinoids, growth hormone, and dietary habits playing roles in its storage and expansion.
- Changes in abdominal fat tissue characteristics, like adipocyte size, inflammation, and how fat is stored and broken down, are closely associated with cardiometabolic risks. Interventions such as lifestyle changes, certain medications, and hormone therapies can help manage fat distribution, although some treatments come with significant side effects.]
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Most liked on X and BlueskyFuentes https://t.co/ZHd7tYE8fI https://t.co/GgU4f5Ibww https://t.co/8ZJWWwIeS4 https://t.co/t0soqWOj7R https://t.co/owPVB7Si0p https://t.co/iWg1BhKyl8. https://t.co/e8PpusTbeG
@FatEmperor Was that a literal question? If so Pathophysiology of Human Visceral Obesity: An Update https://t.co/4QQuwVcziX https://t.co/nDjj1U6mqU
@tednaiman Tchernof A, Després JP. Pathophysiology of human visceral obesity: an update. Physiol Rev. 2013;93(1):359-404.
@Silver65793782 @albemilano È così. Consideri che è il grasso viscerale (intra-addominale) in eccesso. https://t.co/46vU6eWJ9D
El IMC informa acerca del tamaño corporal y tiende a subestimar la grasa corporal, que es la principal causante de las alteraciones metabólicas de la obesidad. de @Medscape https://t.co/m0M7qgKZrx 2013 the American Physiological Society https://t.co/fscyf8Xt5T
All mentions
Understanding Visceral Obesity and Its Health Risks
Visceral obesity involves intraabdominal fat accumulation linked to metabolic issues like insulin resistance and cardiovascular risk. Factors include age, gender, genetics, hormones, and diet. Lifestyle and medical…
Understanding the Risks of Visceral Belly Fat
Visceral obesity involves intraabdominal fat linked to metabolic problems like high triglycerides, inflammation, insulin resistance, and increased cardiometabolic risk. Factors include age, gender, genetics, and…
Understanding the TOFI Phenotype and Fructose's Role in Visceral Fat Formation
This video introduces the TOFI phenotype and explores how fructose induces inflammation and cortisol increase, promoting visceral fat storage and metabolic issues. It highlights the unique impact of fructose on organ…
Diet and Strategies to Reduce Visceral Fat Effectively
Continuing the discussion on visceral fat, this video explores dietary and lifestyle tips for reduction, highlighting effective foods, habits, and interventions to lower abdominal fat and improve metabolic health.
Stress and Hormones Contributing to Persistent Belly Fat
Stress levels and the hormone cortisol may be key factors behind stubborn belly fat, despite diet and exercise efforts. Understanding these connections can help address visceral obesity and its associated health risks.
Understanding Visceral Fat Risks and How to Reduce Abdominal Fat
Visceral fat accumulation links to metabolic issues like insulin resistance and cardiovascular risk. Factors such as age, gender, genetics, and lifestyle influence fat distribution. Learn effective strategies to reduce…
Nutrient Timing: Optimizing Post-Exercise Anabolic Potential
Nutrient timing highlights the importance of protein and carbohydrate intake around exercise sessions to enhance body composition. This video explores recent research on post-exercise nutrition and its impact on muscle…
Optimizing Weight Loss: The Science Behind Nutrient Timing
Nutrient timing focuses on consuming protein and carbohydrates around exercise sessions to enhance body composition. This video reviews evidence-based nutritional recommendations for post-exercise adaptations, providing…
Does Spot Reduction Work for Fat Loss? Scientific Insights
This video explores the myths surrounding spot reduction, showing that targeted abdominal exercises do not significantly reduce fat but improve muscle endurance. Learn proven fat loss strategies for effective results.
In all thy fat gettings, avoid BellyFat AKA android or apple obesity as per Prof Jean Vague's morphology nomenclature: https://t.co/yHzRArXmwN https://t.co/ALmpS74Ao4
Was reading this study on the pathophysiology of visceral fat, and there were a few things that I wanted to highlight (because I'm shady and I'm tired of Black men comparing Black women to other races when it comes to weight.. https://t.co/YPEvVvtZ49
@nntaleb https://t.co/omMTI9cd0m
@Silver65793782 @albemilano È così. Consideri che è il grasso viscerale (intra-addominale) in eccesso. https://t.co/46vU6eWJ9D
@SchubertCalc @theRealDanVCFA @Moralityrevived This review is more than a decade old, but still a good place to start. Tchernof, A., and J.-P. Despres. “Pathophysiology of Human Visceral Obesity: An Update.” Physiological Reviews 93, no. 1 (2013): 359–404. https://t.co/LDGzrN7LBt
RT @ndanmontero: El IMC informa acerca del tamaño corporal y tiende a subestimar la grasa corporal, que es la principal causante de las alt…
El IMC informa acerca del tamaño corporal y tiende a subestimar la grasa corporal, que es la principal causante de las alteraciones metabólicas de la obesidad. de @Medscape https://t.co/m0M7qgKZrx 2013 the American Physiological Society https://t.co/fscyf8Xt5T
@_CynthiaThurlow @Endoftheroast Find section sex hormones… https://t.co/MCWX8aOKXe
@3xtraspicy @DrSeanOMara section E discusses visceral adiposity and cancer https://t.co/9MRYP4BaAm
RT @gerdosi: @FatEmperor @LDLSkeptic @Drlipid @TrueHealthDiag @TuckerGoodrich @daveketo @siobhan_huggins That’s why the indices TG/HDL and…
RT @gerdosi: @FatEmperor @LDLSkeptic @Drlipid @TrueHealthDiag @TuckerGoodrich @daveketo @siobhan_huggins That’s why the indices TG/HDL and…
RT @gerdosi: @FatEmperor @LDLSkeptic @Drlipid @TrueHealthDiag @TuckerGoodrich @daveketo @siobhan_huggins That’s why the indices TG/HDL and…
RT @gerdosi: @FatEmperor @LDLSkeptic @Drlipid @TrueHealthDiag @TuckerGoodrich @daveketo @siobhan_huggins That’s why the indices TG/HDL and…
RT @gerdosi: @FatEmperor @LDLSkeptic @Drlipid @TrueHealthDiag @TuckerGoodrich @daveketo @siobhan_huggins That’s why the indices TG/HDL and…
RT @gerdosi: @FatEmperor @LDLSkeptic @Drlipid @TrueHealthDiag @TuckerGoodrich @daveketo @siobhan_huggins That’s why the indices TG/HDL and…
RT @gerdosi: @FatEmperor @LDLSkeptic @Drlipid @TrueHealthDiag @TuckerGoodrich @daveketo @siobhan_huggins That’s why the indices TG/HDL and…
RT @gerdosi: @FatEmperor @LDLSkeptic @Drlipid @TrueHealthDiag @TuckerGoodrich @daveketo @siobhan_huggins That’s why the indices TG/HDL and…
RT @gerdosi: @FatEmperor @LDLSkeptic @Drlipid @TrueHealthDiag @TuckerGoodrich @daveketo @siobhan_huggins That’s why the indices TG/HDL and…
@FatEmperor @LDLSkeptic @Drlipid @TrueHealthDiag @TuckerGoodrich @daveketo @siobhan_huggins That’s why the indices TG/HDL and TyG usually correlate much better with CVD risk than LDL-C. https://t.co/4QQuwVcziX https://t.co/o8nDuf8uGc
RT @DrMijailTapia: Fuentes https://t.co/ZHd7tYE8fI https://t.co/GgU4f5Ibww https://t.co/8ZJWWwIeS4 https://t.co/t0soqWOj7R https://t.co/owP…
RT @DrMijailTapia: Fuentes https://t.co/ZHd7tYE8fI https://t.co/GgU4f5Ibww https://t.co/8ZJWWwIeS4 https://t.co/t0soqWOj7R https://t.co/owP…
Fuentes https://t.co/ZHd7tYE8fI https://t.co/GgU4f5Ibww https://t.co/8ZJWWwIeS4 https://t.co/t0soqWOj7R https://t.co/owPVB7Si0p https://t.co/iWg1BhKyl8. https://t.co/e8PpusTbeG
Bravo à notre collègue André Tchernof et à Jean-Pierre Després dont l'article Pathophysiology of Human Visceral Obesity: An Update figure parmi le top 100 des articles ayant marqué la revue scientifique Physiological Reviews. 1177 citations ! https://t.co/XrpjjnCFe2
RT @mushawah1: تؤدي الشحوم العميقه او الداخليه بالاحشاء الى الامراض المتنوعه و منها زيادة افراز مواد الالتهابات و الاصابه بالسكري و امراض ا…
RT @mushawah1: تؤدي الشحوم العميقه او الداخليه بالاحشاء الى الامراض المتنوعه و منها زيادة افراز مواد الالتهابات و الاصابه بالسكري و امراض ا…
RT @mushawah1: تؤدي الشحوم العميقه او الداخليه بالاحشاء الى الامراض المتنوعه و منها زيادة افراز مواد الالتهابات و الاصابه بالسكري و امراض ا…
RT @mushawah1: تؤدي الشحوم العميقه او الداخليه بالاحشاء الى الامراض المتنوعه و منها زيادة افراز مواد الالتهابات و الاصابه بالسكري و امراض ا…
RT @mushawah1: تؤدي الشحوم العميقه او الداخليه بالاحشاء الى الامراض المتنوعه و منها زيادة افراز مواد الالتهابات و الاصابه بالسكري و امراض ا…
RT @mushawah1: تؤدي الشحوم العميقه او الداخليه بالاحشاء الى الامراض المتنوعه و منها زيادة افراز مواد الالتهابات و الاصابه بالسكري و امراض ا…
RT @tjavanainen2018: @tednaiman Tchernof A, Després JP. Pathophysiology of human visceral obesity: an update. Physiol Rev. 2013;93(1):359-4…
RT @tjavanainen2018: @tednaiman Tchernof A, Després JP. Pathophysiology of human visceral obesity: an update. Physiol Rev. 2013;93(1):359-4…
@tednaiman Tchernof A, Després JP. Pathophysiology of human visceral obesity: an update. Physiol Rev. 2013;93(1):359-404.
@gerdosi @raphaels7 almost like TZDs “in charge” of dietary fat redistribution? havent seen TZDs used in the context of “T2D remission” or other larger improvements in glycaemic control - must have a look Tchernof (2013) https://t.co/rURfTo6Gjr https://t.co/8fXbZ0YaT4
RT @gerdosi: @FatEmperor Was that a literal question? If so Pathophysiology of Human Visceral Obesity: An Update https://t.co/4QQuwVcziX h…
RT @gerdosi: @FatEmperor Was that a literal question? If so Pathophysiology of Human Visceral Obesity: An Update https://t.co/4QQuwVcziX h…
RT @gerdosi: @FatEmperor Was that a literal question? If so Pathophysiology of Human Visceral Obesity: An Update https://t.co/4QQuwVcziX h…
RT @gerdosi: @FatEmperor Was that a literal question? If so Pathophysiology of Human Visceral Obesity: An Update https://t.co/4QQuwVcziX h…
@FatEmperor Was that a literal question? If so Pathophysiology of Human Visceral Obesity: An Update https://t.co/4QQuwVcziX https://t.co/nDjj1U6mqU
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