Effect of high doses of vitamin D supplementation on dengue virus replication, Toll-like receptor expression, and cytokine profiles on dendritic cells. https://t.co/RMzwx2yvu0
Effect of high doses of vitamin D supplementation on dengue virus replication, Toll-like receptor expression, and cytokine profiles on dendritic cells.
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Nov 2019 – Nov 2020Where it's discussed
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X / Twitter7 posts
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Facebook3 posts
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YouTube2 videos
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AI synopsis
What people are saying
The groups' comments reflect a moderate level of engagement, with mentions of how high doses of vitamin D may influence dengue virus replication and immune responses, such as cytokine changes. The tone remains fairly neutral and informational, focusing on what the study examines rather than advocating for policy change or expressing strong opinions, which explains the neutral impact and interest scores.
The discussions are mostly neutral or mildly supportive, with no strong disagreement expressed about the study.
Participants show some curiosity, especially regarding the potential health implications of vitamin D in dengue, but not overwhelming enthusiasm.
There is moderate engagement, with users referencing the core aspects of the study like immune responses and cytokine modulation, indicating a basic level of thoughtful discussion.
The overall influence appears limited, as the comments suggest interest but do not demonstrate widespread acknowledgment of major implications or calls for action.
Abstract in brief
- The study investigates how high doses of vitamin D supplementation influence dengue virus (DENV) infection and immune responses in dendritic cells, finding that 4000 IU/day of vitamin D reduces DENV infection and modulates immune markers.
- Specifically, higher vitamin D intake decreases susceptibility of dendritic cells to DENV2, reduces the expression of toll-like receptors (TLRs), lowers pro-inflammatory cytokines (IL-12, IL-8), and increases anti-inflammatory cytokine (IL-10).
- These results suggest that vitamin D could potentially enhance innate immunity against DENV infection, but further research is needed to fully understand its role in viral replication and immune regulation.]
Top posts
Most liked on X and BlueskyEffect of high doses of vitamin D supplementation on dengue virus replication, Toll-like receptor expression, and cytokine profiles on dendritic cells | SpringerLink https://t.co/PRzL8SSpj7
Effect of high doses of vitamin D supplementation on dengue virus replication, Toll-like receptor expression, and cytokine profiles on dendritic cells https://t.co/yrDn3an4dL
RT @EurekaMag: Effect of high doses of vitamin D supplementation on dengue virus replication, Toll-like receptor expression, and cytokine p…
RT @EurekaMag: Effect of high doses of vitamin D supplementation on dengue virus replication, Toll-like receptor expression, and cytokine p…
RT @DCell_papers: Effect of high doses of vitamin D supplementation on dengue virus replication, Toll-like receptor expression, and cytokin…
All mentions
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RT @EurekaMag: Effect of high doses of vitamin D supplementation on dengue virus replication, Toll-like receptor expression, and cytokine p…
RT @EurekaMag: Effect of high doses of vitamin D supplementation on dengue virus replication, Toll-like receptor expression, and cytokine p…
Effect of high doses of vitamin D supplementation on dengue virus replication, Toll-like receptor expression, and cytokine profiles on dendritic cells https://t.co/yrDn3an4dL
RT @KJuankhernandez: Effect of high doses of vitamin D supplementation on dengue virus replication, Toll-like receptor expression, and cyto…
Effect of high doses of vitamin D supplementation on dengue virus replication, Toll-like receptor expression, and cytokine profiles on dendritic cells | SpringerLink https://t.co/PRzL8SSpj7
RT @DCell_papers: Effect of high doses of vitamin D supplementation on dengue virus replication, Toll-like receptor expression, and cytokin…
Effect of high doses of vitamin D supplementation on dengue virus replication, Toll-like receptor expression, and cytokine profiles on dendritic cells. https://t.co/RMzwx2yvu0
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