Publications by authors named "Marek Marcinkiewicz"

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Role of saliva in esophageal defense: implications in patients with nonerosive reflux disease.
Harathi Yandrapu, Marek Marcinkiewicz, Cezary Poplawski, Kyung Han, Tomasz Zbroch

Am J Med Sci· May 2015

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A distinct salivary secretory response mediated by the esophago-salivary reflex in patients with Barrett's esophagus: its potential pathogenetic implications.
Harathi Yandrapu, Marek Marcinkiewicz, Cezary Poplawski, Zbigniew Namiot, Tomasz Zbroch

Adv Med Sci· September 2014

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Ileal Immunoglobulin Binding by the Neonatal Fc Receptor: A Previously Unrecognized Mechanism of Protection in the Neonatal Rat Model of Necrotizing Enterocolitis?
Jonathan R Swanson, Tamas Jilling, Jing Lu, Jessica B Landseadel, Marek Marcinkiewicz

EJ Neonatol Res· March 2011

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Exercise and obesity.
Douglas M Okay, Paul V Jackson, Marek Marcinkiewicz, M Novella Papino

Prim Care· June 2009

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A role for plasmin in platelet aggregation: differential regulation of IGF release from IGF-IGFBP complexes?
Marek Marcinkiewicz, Phillip V Gordon

Growth Horm IGF Res· August 2008

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An analysis of IGFBP evolution.
Phillip V Gordon, Marek Marcinkiewicz

Growth Horm IGF Res· August 2008

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A neonatal mouse model of intestinal perforation: investigating the harmful synergism between glucocorticoids and indomethacin.
Phillip V Gordon, Andrew C Herman, Marek Marcinkiewicz, Benjamin M Gaston, Victor E Laubach

J Pediatr Gastroenterol Nutr· November 2007

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Shiga toxin 2 and lipopolysaccharide induce human microvascular endothelial cells to release chemokines and factors that stimulate platelet function.
Fadila Guessous, Marek Marcinkiewicz, Renata Polanowska-Grabowska, Sudawadee Kongkhum, Daniel Heatherly

Infect Immun· December 2005

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Shiga toxin 2 and lipopolysaccharide cause monocytic THP-1 cells to release factors which activate platelet function.
Fadila Guessous, Marek Marcinkiewicz, Renata Polanowska-Grabowska, Tiffany R Keepers, Tom Obrig

Thromb Haemost· November 2005

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LDL oxidized by hypochlorous acid causes irreversible platelet aggregation when combined with low levels of ADP, thrombin, epinephrine, or macrophage-derived chemokine (CCL22).
Leon G Coleman, Renata K Polanowska-Grabowska, Marek Marcinkiewicz, Adrian R L Gear

Blood· July 2004

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