Publications by authors named "Mary Wohltmann"

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Adipocytes are dispensable in shaping the ovarian cancer tumor microenvironment in the omentum.
Rachel L Mintz, Jichang Han, Emily G Butka, Alexandre Gallerand, Ayoung Kim, Mary Wohltmann

bioRxiv· October 2025

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Ileitis-associated tertiary lymphoid organs arise at lymphatic valves and impede mesenteric lymph flow in response to tumor necrosis factor.
Rafael S Czepielewski, Emma C Erlich, Emily J Onufer, Shannon Young, Brian T Saunders, Mary Wohltmann

Immunity· December 2021

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Enterically derived high-density lipoprotein restrains liver injury through the portal vein.
Yong-Hyun Han, Emily J Onufer, Li-Hao Huang, Robert W Sprung, W Sean Davidson, Mary Wohltmann

Science· July 2021

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Limited proliferation capacity of aortic intima resident macrophages requires monocyte recruitment for atherosclerotic plaque progression.
Jesse W Williams, Konstantin Zaitsev, Ki-Wook Kim, Stoyan Ivanov, Brian T Saunders, Mary Wohltmann

Nat Immunol· October 2020

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Metabolic Effects of Selective Deletion of Group VIA Phospholipase A from Macrophages or Pancreatic Islet Beta-Cells.
John Turk, Haowei Song, Mary Wohltmann, Cheryl Frankfater, Xiaoyong Lei

Biomolecules· October 2020

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B Cell-Mediated Antigen Presentation through MHC Class II Is Dispensable for Atherosclerosis Progression.
Jesse W Williams, Andrew Elvington, Skyler Kessler, Mary Wohltmann, Gregory F Wu

Immunohorizons· January 2019

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Polarization of Macrophages toward M2 Phenotype Is Favored by Reduction in iPLA2β (Group VIA Phospholipase A2).
Jason W Ashley, William D Hancock, Alexander J Nelson, Robert N Bone, Hubert M Tse, Mary Wohltmann

J Biol Chem· October 2016

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Group VIA phospholipase A2 mitigates palmitate-induced β-cell mitochondrial injury and apoptosis.
Haowei Song, Mary Wohltmann, Min Tan, Jack H Ladenson, John Turk

J Biol Chem· May 2014

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Genetic modulation of islet β-cell iPLA₂β expression provides evidence for its impact on β-cell apoptosis and autophagy.
Xiaoyong Lei, Robert N Bone, Tomader Ali, Mary Wohltmann, Ying Gai

Islets· November 2013

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Group VIB Phospholipase A(2) promotes proliferation of INS-1 insulinoma cells and attenuates lipid peroxidation and apoptosis induced by inflammatory cytokines and oxidant agents.
Shunzhong Bao, Haowei Song, Min Tan, Mary Wohltmann, Jack H Ladenson

Oxid Med Cell Longev· April 2013

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Disturbed brain phospholipid and docosahexaenoic acid metabolism in calcium-independent phospholipase A(2)-VIA (iPLA(2)β)-knockout mice.
Yewon Cheon, Hyung-Wook Kim, Miki Igarashi, Hiren R Modi, Lisa Chang, Mary Wohltmann

Biochim Biophys Acta· September 2012

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Group VIA PLA2 (iPLA2β) is activated upstream of p38 mitogen-activated protein kinase (MAPK) in pancreatic islet β-cell signaling.
Haowei Song, Mary Wohltmann, Min Tan, Shunzhong Bao, Jack H Ladenson

J Biol Chem· February 2012

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Imaging decreased brain docosahexaenoic acid metabolism and signaling in iPLA(2)β (VIA)-deficient mice.
Mireille Basselin, Angelo O Rosa, Epolia Ramadan, Yewon Cheon, Lisa Chang, Mary Wohltmann

J Lipid Res· November 2010

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Effects of endoplasmic reticulum stress on group VIA phospholipase A2 in beta cells include tyrosine phosphorylation and increased association with calnexin.
Haowei Song, Henry Rohrs, Min Tan, Mary Wohltmann, Jack H Ladenson

J Biol Chem· October 2010

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Mice deficient in group VIB phospholipase A2 (iPLA2gamma) exhibit relative resistance to obesity and metabolic abnormalities induced by a Western diet.
Haowei Song, Mary Wohltmann, Shunzhong Bao, Jack H Ladenson, Clay F Semenkovich

Am J Physiol Endocrinol Metab· June 2010

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Age-related changes in bone morphology are accelerated in group VIA phospholipase A2 (iPLA2beta)-null mice.
Sasanka Ramanadham, Kevin E Yarasheski, Matthew J Silva, Mary Wohltmann, Deborah Veis Novack

Am J Pathol· April 2008

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Glucose homeostasis, insulin secretion, and islet phospholipids in mice that overexpress iPLA2beta in pancreatic beta-cells and in iPLA2beta-null mice.
Shunzhong Bao, David A Jacobson, Mary Wohltmann, Alan Bohrer, Wu Jin

Am J Physiol Endocrinol Metab· February 2008

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Disrupted membrane homeostasis and accumulation of ubiquitinated proteins in a mouse model of infantile neuroaxonal dystrophy caused by PLA2G6 mutations.
Ibrahim Malik, John Turk, David J Mancuso, Laura Montier, Mary Wohltmann

Am J Pathol· February 2008

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Attenuated free cholesterol loading-induced apoptosis but preserved phospholipid composition of peritoneal macrophages from mice that do not express group VIA phospholipase A2.
Shunzhong Bao, Yankun Li, Xiaoyong Lei, Mary Wohltmann, Wu Jin

J Biol Chem· September 2007

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Insulin secretory responses and phospholipid composition of pancreatic islets from mice that do not express Group VIA phospholipase A2 and effects of metabolic stress on glucose homeostasis.
Shunzhong Bao, Haowei Song, Mary Wohltmann, Sasanka Ramanadham, Wu Jin

J Biol Chem· July 2006

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Male mice that do not express group VIA phospholipase A2 produce spermatozoa with impaired motility and have greatly reduced fertility.
Shunzhong Bao, David J Miller, Zhongmin Ma, Mary Wohltmann, Grace Eng

J Biol Chem· September 2004

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Delta6-, Stearoyl CoA-, and Delta5-desaturase enzymes are expressed in beta-cells and are altered by increases in exogenous PUFA concentrations.
Sasanka Ramanadham, Sheng Zhang, Zhongmin Ma, Mary Wohltmann, Alan Bohrer

Biochim Biophys Acta· January 2002

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