Publications by authors named "Alister P Funnell"

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Krüppel-like Factor 3 (KLF3/BKLF) Is Required for Widespread Repression of the Inflammatory Modulator Galectin-3 (Lgals3).
Alexander J Knights, Jinfen J Yik, Hanapi Mat Jusoh, Laura J Norton, Alister P W Funnell

J Biol Chem· July 2016

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Erratum to: Repression of chimeric transcripts emanating from endogenous retrotransposons by a sequence-specific transcription factor.
Ka Sin Mak, Jon Burdach, Laura J Norton, Richard Cm Pearson, Merlin Crossley, Alister Pw Funnell

Genome Biol· June 2016

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Directing an artificial zinc finger protein to new targets by fusion to a non-DNA-binding domain.
Wooi F Lim, Jon Burdach, Alister P W Funnell, Richard C M Pearson, Kate G R Quinlan

Nucleic Acids Res· April 2016

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Transcription factors LRF and BCL11A independently repress expression of fetal hemoglobin.
Takeshi Masuda, Xin Wang, Manami Maeda, Matthew C Canver, Falak Sher, Alister P W Funnell

Science· January 2016

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2p15-p16.1 microdeletions encompassing and proximal to BCL11A are associated with elevated HbF in addition to neurologic impairment.
Alister P W Funnell, Paolo Prontera, Valentina Ottaviani, Maria Piccione, Antonino Giambona

Blood· July 2015

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Editing the genome to introduce a beneficial naturally occurring mutation associated with increased fetal globin.
Beeke Wienert, Alister P W Funnell, Laura J Norton, Richard C M Pearson, Lorna E Wilkinson-White

Nat Commun· May 2015

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Phosphorylation of Krüppel-like factor 3 (KLF3/BKLF) and C-terminal binding protein 2 (CtBP2) by homeodomain-interacting protein kinase 2 (HIPK2) modulates KLF3 DNA binding and activity.
Vitri Dewi, Alister Kwok, Stella Lee, Ming Min Lee, Yee Mun Tan, Alister P W Funnell

J Biol Chem· March 2015

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Multi-species, multi-transcription factor binding highlights conserved control of tissue-specific biological pathways.
Benoit Ballester, Alejandra Medina-Rivera, Dominic Schmidt, Mar Gonzàlez-Porta, Matthew Carlucci, Alister P W Funnell

Elife· October 2014

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Differential regulation of the α-globin locus by Krüppel-like Factor 3 in erythroid and non-erythroid cells.
Alister P W Funnell, Douglas Vernimmen, Wooi F Lim, Ka Sin Mak, Beeke Wienert

BMC Mol Biol· May 2014

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Adipokines and insulin action: A sensitive issue.
Alexander J Knights, Alister Pw Funnell, Richard Cm Pearson, Merlin Crossley, Kim S Bell-Anderson

Adipocyte· April 2014

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Repression of chimeric transcripts emanating from endogenous retrotransposons by a sequence-specific transcription factor.
Ka Sin Mak, Jon Burdach, Laura J Norton, Richard C M Pearson, Merlin Crossley, Alister P W Funnell

Genome Biol· April 2014

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ENU-induced mutation in the DNA-binding domain of KLF3 reveals important roles for KLF3 in cardiovascular development and function in mice.
Lois Kelsey, Ann M Flenniken, Dawei Qu, Alister P W Funnell, Richard Pearson

PLoS Genet· January 2014

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Regions outside the DNA-binding domain are critical for proper in vivo specificity of an archetypal zinc finger transcription factor.
Jon Burdach, Alister P W Funnell, Ka Sin Mak, Crisbel M Artuz, Beeke Wienert

Nucleic Acids Res· January 2014

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Hemophilia B Leyden and once mysterious cis-regulatory mutations.
Alister P W Funnell, Merlin Crossley

Trends Genet· January 2014

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Loss of Krüppel-like factor 3 (KLF3/BKLF) leads to upregulation of the insulin-sensitizing factor adipolin (FAM132A/CTRP12/C1qdc2).
Kim S Bell-Anderson, Alister P Funnell, Helen Williams, Hanapi Mat Jusoh, Tiffany Scully

Diabetes· August 2013

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Generation of mice deficient in both KLF3/BKLF and KLF8 reveals a genetic interaction and a role for these factors in embryonic globin gene silencing.
Alister P W Funnell, Ka Sin Mak, Natalie A Twine, Gregory J Pelka, Laura J Norton

Mol Cell Biol· August 2013

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A CpG mutational hotspot in a ONECUT binding site accounts for the prevalent variant of hemophilia B Leyden.
Alister P W Funnell, Michael D Wilson, Benoit Ballester, Ka Sin Mak, Jon Burdach

Am J Hum Genet· March 2013

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Homo- and heterodimerization in transcriptional regulation.
Alister P W Funnell, Merlin Crossley

Adv Exp Med Biol· October 2012

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The CACCC-binding protein KLF3/BKLF represses a subset of KLF1/EKLF target genes and is required for proper erythroid maturation in vivo.
Alister P W Funnell, Laura J Norton, Ka Sin Mak, Jon Burdach, Crisbel M Artuz

Mol Cell Biol· August 2012

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Holding Tight: Cell Junctions and Cancer Spread.
Alexander J Knights, Alister P W Funnell, Merlin Crossley, Richard C M Pearson

Trends Cancer Res· January 2012

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Cellular Reprogramming toward the Erythroid Lineage.
Laura J Norton, Alister P W Funnell, Richard C M Pearson, Merlin Crossley

Int J Cell Biol· November 2011

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PU.1 and Haematopoietic Cell Fate: Dosage Matters.
Ka Sin Mak, Alister P W Funnell, Richard C M Pearson, Merlin Crossley

Int J Cell Biol· November 2011

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Impaired B cell development in the absence of Krüppel-like factor 3.
Thi Thanh Vu, Dominique Gatto, Vivian Turner, Alister P W Funnell, Ka Sin Mak

J Immunol· November 2011

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The mammalian zinc finger transcription factor Krüppel-like factor 3 (KLF3/BKLF).
Richard C M Pearson, Alister P W Funnell, Merlin Crossley

IUBMB Life· February 2011

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A network of Krüppel-like Factors (Klfs). Klf8 is repressed by Klf3 and activated by Klf1 in vivo.
Sally A Eaton, Alister P W Funnell, Nancy Sue, Hannah Nicholas, Richard C M Pearson

J Biol Chem· October 2008

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