Publications by authors named "Peter A Bell"

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Altered pattern of proteolysis in rhegmatogenous retinal detachment by mining of N-termini datasets from vitreous humor proteome.
Gabriele Antonio Zingale, Sara Giammaria, Irene Pandino, Luca Placentino, Guido Ripandelli, Peter A Bell

Sci Rep· October 2025

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SpectraSage unveils specific proteolytic patterns of 20S on mono-ubiquitylated Tau proteoforms involved in neurodegeneration.
Gabriele Antonio Zingale, Irene Pandino, Daniele Trivellato, Dario Cavaterra, Francesca Munari, Peter A Bell

Chem Sci· September 2025

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SARS-CoV-2 3CL (main protease) regulates caspase activation of gasdermin-D/E pores leading to secretion and extracellular activity of 3CL.
Peter M Grin, Kaushal Baid, Hugo C R de Jesus, Nedim Kozarac, Peter A Bell

Cell Rep· December 2024

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Optimization of quenched fluorescent peptide substrates of SARS-CoV-2 3CL main protease (Mpro) from proteomic identification of P6-P6' active site specificity.
Hugo Cesar Ramos de Jesus, Nestor Solis, Yoan Machado, Isabel Pablos, Peter A Bell

J Virol· June 2024

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No Substrate Left behind-Mining of Shotgun Proteomics Datasets Rescues Evidence of Proteolysis by SARS-CoV-2 3CL Main Protease.
Peter A Bell, Christopher M Overall

Int J Mol Sci· May 2023

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Integrating knowledge of protein sequence with protein function for the prediction and validation of new MALT1 substrates.
Peter A Bell, Sophia Scheuermann, Florian Renner, Christina L Pan, Henry Y Lu

Comput Struct Biotechnol J· August 2022

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Mechanistic understanding of the combined immunodeficiency in complete human CARD11 deficiency.
Henry Y Lu, Mehul Sharma, Ashish A Sharma, Atilano Lacson, Ashley Szpurko, Peter A Bell

J Allergy Clin Immunol· December 2021

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Mechanistic insights into COVID-19 by global analysis of the SARS-CoV-2 3CL substrate degradome.
Isabel Pablos, Yoan Machado, Hugo C Ramos de Jesus, Yasir Mohamud, Reinhild Kappelhoff, Peter A Bell

Cell Rep· October 2021

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Moonlighting matrix metalloproteinase substrates: Enhancement of proinflammatory functions of extracellular tyrosyl-tRNA synthetase upon cleavage.
Parker G Jobin, Nestor Solis, Yoan Machado, Peter A Bell, Simran K Rai

J Biol Chem· February 2020

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Proteomic and N-Terminomic TAILS Analyses of Human Alveolar Bone Proteins: Improved Protein Extraction Methodology and LysargiNase Digestion Strategies Increase Proteome Coverage and Missing Protein Identification.
Peter A Bell, Nestor Solis, Jayachandran N Kizhakkedathu, Ian Matthew, Christopher M Overall

J Proteome Res· December 2019

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Matrix metalloproteinases inactivate the proinflammatory functions of secreted moonlighting tryptophanyl-tRNA synthetase.
Parker G Jobin, Nestor Solis, Yoan Machado, Peter A Bell, Nam Hoon Kwon

J Biol Chem· August 2019

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New therapeutic targets in rare genetic skeletal diseases.
Michael D Briggs, Peter A Bell, Michael J Wright, Katarzyna A Pirog

Expert Opin Orphan Drugs· October 2015

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The utility of mouse models to provide information regarding the pathomolecular mechanisms in human genetic skeletal diseases: The emerging role of endoplasmic reticulum stress (Review).
Michael D Briggs, Peter A Bell, Katarzyna A Pirog

Int J Mol Med· June 2015

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Abnormal chondrocyte apoptosis in the cartilage growth plate is influenced by genetic background and deletion of CHOP in a targeted mouse model of pseudoachondroplasia.
Katarzyna A Piróg, Andreja Irman, Siobhan Young, Poonam Halai, Peter A Bell

PLoS One· January 2015

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Genotype to phenotype correlations in cartilage oligomeric matrix protein associated chondrodysplasias.
Michael D Briggs, Joanne Brock, Simon C Ramsden, Peter A Bell

Eur J Hum Genet· November 2014

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Armet/Manf and Creld2 are components of a specialized ER stress response provoked by inappropriate formation of disulphide bonds: implications for genetic skeletal diseases.
Claire L Hartley, Sarah Edwards, Lorna Mullan, Peter A Bell, Maryline Fresquet

Hum Mol Genet· December 2013

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A novel transgenic mouse model of growth plate dysplasia reveals that decreased chondrocyte proliferation due to chronic ER stress is a key factor in reduced bone growth.
Benedetta Gualeni, M Helen Rajpar, Aaron Kellogg, Peter A Bell, Peter Arvan

Dis Model Mech· November 2013

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Analysis of the cartilage proteome from three different mouse models of genetic skeletal diseases reveals common and discrete disease signatures.
Peter A Bell, Raimund Wagener, Frank Zaucke, Manuel Koch, Julian Selley

Biol Open· August 2013

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Loss of matrilin 1 does not exacerbate the skeletal phenotype in a mouse model of multiple epiphyseal dysplasia caused by a Matn3 V194D mutation.
Peter A Bell, Katarzyna A Piróg, Maryline Fresquet, David J Thornton, Raymond P Boot-Handford

Arthritis Rheum· May 2012

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An unfolded protein response is the initial cellular response to the expression of mutant matrilin-3 in a mouse model of multiple epiphyseal dysplasia.
Seema Nundlall, M Helen Rajpar, Peter A Bell, Christopher Clowes, Leo A H Zeeff

Cell Stress Chaperones· November 2010

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Quantitation of proteins using a dye-metal-based colorimetric protein assay.
Babu S Antharavally, Krishna A Mallia, Priya Rangaraj, Paul Haney, Peter A Bell

Anal Biochem· February 2009

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What do we really know about neurological misdiagnosis in the emergency department?
Janice Wachtler, Peter A Bell, Thomas Brabson

Mayo Clin Proc· February 2008

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Solid-phase biotinylation of antibodies.
Elizabeth Strachan, A Krishna Mallia, Joanna M Cox, Babu Antharavally, Surbhi Desai, Peter A Bell

J Mol Recognit· December 2004

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A high-affinity reversible protein stain for Western blots.
Babu S Antharavally, Brad Carter, Peter A Bell, A Krishna Mallia

Anal Biochem· June 2004

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Recent developments in medicine and law.
Melissa A Wojtylak, Kenneth C Bartholomew, Peter A Bell, Michael J Farrell, Melissa K Ferrell

Tort Trial Insur Pract Law J· April 2004

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